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Cloning and sequencing of a new 15-hydroxyprostaglandin dehydrogenase related mRNA
R Delage-Mourroux1, F Pichaud, J L Frendo
1INSERM U 349, Hôpital Lariboisière, Paris, France.
Abstract:
NAD+-dependent 15-hydroxyprostaglandin dehydrogenase (type-I 15-PGDH) inactivates prostaglandins. We recently reported an mRNA sequence coding for a predicted isomer (PGDH(rI)) of this enzyme. The TT cell line, derived from medullary thyroid carcinoma (MTC), expresses mRNAs for both isomers. We report here the expression by TT cells and MTC of a third 15-PGDH related mRNA (PGDH(rII)), 241 nt shorter than type-I 15-PGDH. RNase protection assays confirmed that TT cells expressed this mRNA (PGDH(rII)). Thus different splicing patterns could be involved in the post-transcriptional regulation of type-I 15-PGDH gene in MTC.
Insights
Researchers identified a new mRNA variant, PGDH(rII), related to the prostaglandin-inactivating enzyme 15-PGDH. This finding in medullary thyroid carcinoma suggests alternative splicing regulates 15-PGDH gene expression.
Area of Science:
- Biochemistry
- Molecular Biology
- Oncology
Background:
- NAD+-dependent 15-hydroxyprostaglandin dehydrogenase (15-PGDH) is crucial for prostaglandin inactivation.
- Previous work identified an mRNA isomer (PGDH(rI)) of 15-PGDH.
- Medullary thyroid carcinoma (MTC) cell lines express mRNAs for both known 15-PGDH isomers.
Purpose of the Study:
- To investigate the expression of novel 15-PGDH-related mRNAs in MTC.
- To characterize a newly identified mRNA variant in TT cells and MTC.
Main Methods:
- RNase protection assays were utilized to detect and confirm mRNA expression.
- Analysis of mRNA sequences to identify variants and their characteristics.
Main Results:
- A third 15-PGDH-related mRNA, termed PGDH(rII), was identified.
- PGDH(rII) is 241 nucleotides shorter than the type-I 15-PGDH mRNA.
- TT cells and MTC express the PGDH(rII) mRNA.
Conclusions:
- The expression of PGDH(rII) in MTC suggests a more complex regulation of 15-PGDH.
- Alternative splicing patterns are likely involved in the post-transcriptional regulation of the type-I 15-PGDH gene in MTC.