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Complex pattern of inhibition by Mg2+ of exocytosis from permeabilised eosinophils

K Y Larbi1, B D Gomperts

  • 1Department of Physiology, University College London, UK.

Cell Calcium
|March 1, 1997
PubMed

Insights

Magnesium ions (Mg2+) inhibit exocytotic secretion in eosinophils. This inhibition may involve GTP-binding proteins, with Mg2+ playing a complex, biphasic role dependent on ATP presence.

Area of Science:

  • Cell Biology
  • Immunology
  • Biochemistry

Background:

  • Exocytotic secretion is crucial for eosinophil function.
  • Calcium (Ca2+) and GTP-binding proteins (G proteins) are key regulators of secretion.
  • The role of magnesium ions (Mg2+) in eosinophil secretion is not fully understood.

Purpose of the Study:

  • To investigate the inhibitory effects of Mg2+ on exocytotic secretion in permeabilized eosinophils.
  • To elucidate the mechanisms by which Mg2+ influences secretion stimulated by Ca2+ and GTPγS, with and without ATP.

Main Methods:

  • Utilized permeabilized eosinophils stimulated with Ca2+ and GTPγS.
  • Assessed the release of specific enzymes (aryl sulphatase, hexosaminidase, peroxidase) as indicators of secretion.
  • Varied concentrations of Mg2+ and ATP to determine dose-dependent effects and identify IC50 values.

Main Results:

  • Mg2+ inhibited the release of aryl sulphatase, hexosaminidase, and peroxidase.
  • Mg2+ inhibition did not appear to compete with Ca2+-binding sites.
  • Mg2+ showed an inverse relationship with GTPγS efficacy, suggesting interaction with G protein-dependent events.
  • Mg2+ exhibited biphasic effects when ATP was present and Ca2+ was absent, initially promoting and then inhibiting secretion.

Conclusions:

  • Mg2+ plays a complex regulatory role in eosinophil exocytosis.
  • Inhibition by Mg2+ likely involves G protein pathways rather than direct competition with Ca2+.
  • The presence of ATP modulates Mg2+ inhibition, highlighting the intricate interplay of ions and cofactors in secretion.

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