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Verbal conditioning of angina pectoris during exercise testing
The American Journal of Cardiology
|October 1, 1977
Summary
Psychological factors like verbal conditioning may trigger angina pectoris in patients with coronary heart disease. This study shows pain and S-T segment depression occurred at a specific count, regardless of actual exercise intensity.
Area of Science:
- Cardiology
- Psychosomatic Medicine
- Exercise Physiology
Background:
- Coronary heart disease (CHD) management often involves exercise testing to assess functional capacity and ischemia.
- The Master two-step test is a common exercise protocol used to evaluate patients with suspected or known CHD.
- Patient perception and reporting of symptoms during exercise are crucial for test interpretation.
Observation:
- A patient with advanced coronary heart disease experienced angina pectoris and significant S-T segment depression during a Master two-step test.
- These symptoms consistently appeared at the 44th crossing, irrespective of the actual exercise duration due to a miscount.
- When the exercise count was accurate and less intense, symptoms did not manifest at the same perceived exertion level.
Findings:
- The patient's angina and S-T segment depression were consistently elicited at a specific perceived exertion level (44 crossings), suggesting a potential role for psychological factors.
- The absence of symptoms at a lower, accurately counted exercise level indicates that the physical exertion alone may not have been the sole trigger.
- This suggests that verbal conditioning or the expectation of symptoms at a certain point may have influenced the physiological response.
Implications:
- Explores the potential for verbal conditioning to provoke or exacerbate angina pectoris in individuals with established coronary heart disease.
- Highlights the importance of considering the psychological component in the manifestation of ischemic symptoms during exercise.
- Suggests that patient perception and cognitive factors may play a more significant role in exercise-induced angina than previously understood.