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Conjugative mobilization of a vancomycin resistance plasmid by a putative Enterococcus faecium sex pheromone response

M P Heaton1, S Handwerger

  • 1Laboratory of Microbiology, Rockefeller University, New York, New York 10021, USA.

Microbial Drug Resistance (Larchmont, N.Y.)
|July 1, 1995
PubMed

Insights

Horizontal gene transfer aids vancomycin resistance spread. A specific Enterococcus faecium plasmid (pHKK703) mobilizes another (pHKK702) carrying resistance genes, requiring donor cell recombination for efficient transfer.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Epidemiological data suggest horizontal gene transfer (HGT) is crucial for vancomycin resistance dissemination.
  • Vancomycin resistance in Enterococcus faecium is a significant public health concern.
  • Understanding the mechanisms of resistance gene transfer is vital for combating antimicrobial resistance.

Purpose of the Study:

  • To investigate the plasmid-mediated transfer of vancomycin resistance (Vmr) from Enterococcus faecium.
  • To characterize the plasmids involved in the conjugative mobilization of Vmr.
  • To elucidate the role of donor cell recombination in Vmr transfer.

Main Methods:

  • Filter mating and liquid culture assays were used to assess Vmr transfer between Enterococcus strains.
  • Plasmid content analysis of transconjugants was performed using hybridization techniques.
  • Recombination-deficient recipient strains were employed to evaluate the role of recombination in transfer.

Main Results:

  • 14 out of 21 VanA Enterococcus faecium isolates transferred Vmr to Enterococcus faecalis JH2-2.
  • Two plasmids, pHKK702 (41-kb) and pHKK703 (55-kb), were identified in efficient Vmr transconjugants.
  • pHKK702 contains a Tn1546-like element and an ermB-hybridizing element; pHKK703 contains sex pheromone response genes.
  • Efficient Vmr transfer required both plasmids; transfer was significantly reduced in recombination-deficient recipients.

Conclusions:

  • The plasmid pHKK703 acts as a conjugative mobilizing plasmid for pHKK702, which carries the vancomycin resistance transposon.
  • Donor-mediated recombination proficiency appears to be a significant factor in the mobilization of pHKK702 by pHKK703.
  • This study demonstrates the conjugative mobilization of a Tn1546-related element by a pheromone-responsive plasmid.

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