Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

How smoking affects blood pressure

P Omvik1

  • 1Medical Department A, University of Bergen, School of Medicine, Haukeland Hospital, Norway.

Blood Pressure
|March 1, 1996
PubMed
Summary
This summary is machine-generated.

Nicotine in tobacco smoking acutely raises blood pressure and heart rate, contributing to addiction. However, long-term smokers may have lower blood pressure, necessitating careful antihypertensive treatment initiation during smoking cessation programs.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Lowering Albuminuria-Does It Lower the Cardiovascular Risk?: Reduction in Albuminuria Translates to Reduction in Cardiovascular Events in Hypertensive Patients: Losartan Intervention for Endpoint Reduction in Hypertension Study. Hypertension 45: 198-202, 2005.

Journal of the American Society of Nephrology : JASN·2023
Same author

Reducing Microalbuminuria-Does It Lower Cardiovascular Risk?: Reduction in Albuminuria Translates to Reduction in Cardiovascular Events in Hypertensive Patients: Losartan Intervention for Endpoint Reduction in Hypertension Study. Hypertension 45: 198-202, 2005.

Journal of the American Society of Nephrology : JASN·2023
Same author

Impact of diabetes on treatment-induced changes in left ventricular structure and function in hypertensive patients with left ventricular hypertrophy. The LIFE study.

Nutrition, metabolism, and cardiovascular diseases : NMCD·2009
Same author

Alcohol consumption and cardiovascular risk in hypertensives with left ventricular hypertrophy: the LIFE study.

Journal of human hypertension·2004
Same author

A retrospective analysis comparing the costs and cost effectiveness of amlodipine and enalapril in the treatment of hypertension.

Managed care interface·2001
Same author

Renal hemodynamic effects of captopril and doxazosin during slight physical activity in hypertensive patients with type-1 diabetes mellitus.

Kidney & blood pressure research·2001

Area of Science:

  • Cardiovascular Physiology
  • Pharmacology of Nicotine
  • Addiction Research

Background:

  • The addictive nature of smoking is not fully understood, with hemodynamic effects potentially playing a role.
  • Tobacco smoking is known to acutely increase blood pressure and heart rate, primarily due to nicotine.
  • Understanding these physiological responses is crucial for managing cardiovascular health in smokers.

Purpose of the Study:

  • To explore the contribution of hemodynamic effects of tobacco smoking to nicotine addiction.
  • To investigate the immediate physiological responses to smoking, including blood pressure and heart rate changes.
  • To examine the interaction between smoking, antihypertensive medications, and blood pressure regulation.

Main Methods:

  • Review of existing knowledge on the hemodynamic effects of tobacco smoking.

Related Experiment Videos

  • Analysis of the immediate physiological responses during smoking, distinguishing nicotine's role.
  • Examination of clinical observations regarding antihypertensive medication efficacy in smokers.

    • Main Results:

    • Nicotine acutely increases blood pressure via enhanced cardiac output and peripheral vascular resistance, preceding catecholamine release.
    • Beta-blocker efficacy may be reduced in smokers, while alpha-receptor blockers appear to maintain antihypertensive effects.
    • Epidemiological studies paradoxically show lower blood pressure in smokers compared to non-smokers, despite acute increases.

    Conclusions:

    • Hemodynamic changes induced by nicotine are significant contributors to smoking habituation.
    • Smoking cessation programs should consider the potential for post-cessation blood pressure increases and not delay antihypertensive treatment.
    • The interaction between smoking and cardiovascular medications requires careful consideration for effective patient management.