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The RLF-M component of the replication licensing system forms complexes containing all six MCM/P1 polypeptides

P Thömmes1, Y Kubota, H Takisawa

  • 1ICRF Clare Hall Laboratories, Potters Bar, Herts, UK.

The EMBO Journal
|June 2, 1997
PubMed

Insights

Replication licensing factor-M (RLF-M) requires all six MCM/P1 proteins (XMcm2-XMcm7) to prevent DNA re-replication. These proteins form a complex that binds chromatin before S-phase, indicating their necessity for replication licensing.

Area of Science:

  • Molecular Biology
  • Cell Cycle Regulation
  • DNA Replication

Background:

  • Replication licensing factor (RLF) prevents DNA re-replication within a single cell cycle.
  • RLF has been previously separated into RLF-M and RLF-B components.

Purpose of the Study:

  • To elucidate the composition and function of Xenopus RLF-M.
  • To determine the role of MCM/P1 proteins in replication licensing.

Main Methods:

  • Glycerol gradient and gel filtration to determine complex molecular weight.
  • Co-immunoprecipitation using anti-XMcm3 antibody.
  • Chromatin binding assays before and during S-phase.

Main Results:

  • Xenopus RLF-M comprises all six MCM/P1 proteins (XMcm2-XMcm7) as a ~400 kDa complex.
  • Fractionation yielded sub-components lacking RLF-M activity, highlighting the necessity of the complete complex.
  • All six MCM/P1 proteins bind chromatin synchronously before S-phase and are displaced during S-phase progression.

Conclusions:

  • The complete complex of six MCM/P1 proteins is essential for replication licensing in Xenopus.
  • MCM/P1 protein complex assembly and chromatin binding dynamics are critical for cell cycle control of DNA replication.

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