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Interaction of Fas(Apo-1/CD95) with proteins implicated in the ubiquitination pathway

K Becker1, P Schneider, K Hofmann

  • 1Institute of Biochemistry, University of Lausanne, Epalinges, Switzerland.

FEBS Letters
|July 21, 1997
PubMed

Insights

The ubiquitin-conjugating enzyme UBC9 binds to Fas, a receptor that triggers apoptosis. This discovery suggests ubiquitination pathway proteins may regulate Fas signaling, impacting programmed cell death.

Area of Science:

  • Cell biology
  • Molecular biology
  • Biochemistry

Background:

  • Fas (Apo-1/CD95) is a tumor necrosis factor receptor family member that initiates apoptosis upon Fas ligand binding.
  • Activated Fas recruits intracellular proteins to relay the apoptotic death signal.
  • Understanding Fas-associated proteins is crucial for elucidating apoptosis regulation.

Purpose of the Study:

  • To identify proteins that associate with Fas using a yeast two-hybrid screen.
  • To investigate the role of ubiquitination pathway components in Fas-mediated apoptosis.
  • To characterize the interaction between UBC9 and Fas.

Main Methods:

  • Yeast two-hybrid screening to identify Fas-binding proteins.
  • In vivo co-expression and binding assays in HeLa cells.
  • Sequence analysis of Fas-associated Factor 1 (FAF1).

Main Results:

  • The ubiquitin-conjugating enzyme UBC9 was identified as a Fas-associated protein.
  • UBC9 binds to Fas at the junction of its death domain and membrane-proximal region.
  • This UBC9-Fas interaction was confirmed in vivo in HeLa cells.
  • Fas-associated Factor 1 (FAF1) exhibits sequence similarity to ubiquitin.

Conclusions:

  • The ubiquitin-conjugating enzyme UBC9 directly interacts with Fas.
  • Proteins involved in the ubiquitination pathway, such as UBC9, may play a regulatory role in Fas-mediated apoptosis.
  • These findings highlight a potential link between ubiquitination and the modulation of programmed cell death pathways.

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