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Plasma total homocysteine concentrations in epileptic patients taking anticonvulsants

H Ono1, A Sakamoto, T Eguchi

  • 1Department of Pediatrics, Hiroshima University School of Medicine, Hiroshima, Japan.

Insights

Measuring serum folate (FA) and plasma total homocysteine (tHcy) can help prevent thrombosis in epileptic patients on long-term anticonvulsant therapy. Low FA levels correlate with high tHcy, especially in older patients.

Area of Science:

  • Clinical Chemistry
  • Neurology
  • Nutritional Biochemistry

Background:

  • Epileptic patients on anticonvulsant drugs often exhibit altered homocysteine and folate levels.
  • Hyperhomocysteinemia is a risk factor for thrombosis.
  • Anticonvulsant medications can interfere with folate metabolism.

Purpose of the Study:

  • To investigate the relationship between serum folate (FA) and plasma total homocysteine (tHcy) in epileptic patients on anticonvulsant therapy.
  • To assess the clinical significance of FA deficiency and hyperhomocysteinemia in this patient group.

Main Methods:

  • Cross-sectional study involving 130 epileptic patients treated with anticonvulsants.
  • Measurement of plasma total homocysteine (tHcy) and serum folate (FA) concentrations.
  • Comparison of tHcy levels with control subjects and correlation analysis between FA and tHcy.

Main Results:

  • A significant inverse correlation was observed between FA and tHcy concentrations, more pronounced in older patients (≥15 years).
  • Four patients (3.1%) were identified as folate-deficient, all with long-term (>7 years) multiple anticonvulsant treatment and elevated tHcy levels.
  • Two patients with extremely high tHcy and subnormal methionine normalized after folate therapy, with tHcy decreasing to control levels.

Conclusions:

  • Monitoring FA and tHcy levels is valuable for preventing thrombosis in epileptic patients on anticonvulsants.
  • Long-term treatment with multiple anticonvulsants increases the risk of folate deficiency and hyperhomocysteinemia.
  • Early detection and intervention with folate therapy can mitigate hyperhomocysteinemia-associated risks.

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