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Related Experiment Videos

The adynamic terminal ureteral segment

A M Singer, J F Glenn

    The Journal of Urology
    |December 1, 1977
    PubMed
    Summary
    This summary is machine-generated.

    Hydroureteronephrosis can persist after bladder outlet obstruction treatment due to ureteral wall damage. This condition, characterized by a weakened distal ureteral segment, can lead to ongoing upper tract dilation and deterioration.

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    Area of Science:

    • Urology
    • Nephrology
    • Pathology

    Background:

    • Bladder outlet obstruction (BOO) commonly leads to upper urinary tract changes.
    • Successful correction of BOO is typically expected to resolve associated hydroureteronephrosis.

    Observation:

    • A case of hydroureteronephrosis was observed.
    • The condition was linked to an adynamic, transmural distal ureteral segment.
    • A thickened bladder wall, secondary to BOO, was also noted.

    Findings:

    • Histological examination revealed loss of intrinsic musculature in the terminal ureteral segment.
    • Fibrosis was present in the affected distal ureteral segment.
    • These findings indicate intrinsic ureteral wall pathology.

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    Implications:

    • Upper tract dilatation and deterioration may continue despite successful BOO correction.
    • Intrinsic ureteral wall disease can be a cause of persistent hydroureteronephrosis.
    • This highlights the need for careful evaluation of the ureteral wall in managing BOO complications.