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Coronary angiographic characteristics of patients with permanent artificial pacemakers

M Mosseri1, T Izak, S Rosenheck

  • 1Cardiology Department, Hadassah Hebrew University Medical Center, Jerusalem, Israel. mosseri@cc.huji.ac.il

Circulation
|August 5, 1997
PubMed

Insights

Severe cardiac conduction disturbances requiring pacemakers are linked to specific coronary artery disease patterns. This includes compromised blood flow to septal branches and right coronary artery lesions, not just diffuse atherosclerosis.

Area of Science:

  • Cardiology
  • Interventional Cardiology
  • Cardiac Electrophysiology

Background:

  • The etiology of severe cardiac conduction disturbances is frequently unclear.
  • This study investigated the role of coronary atherosclerotic disease in patients with permanent pacemakers.

Purpose of the Study:

  • To determine the extent of coronary atherosclerotic disease responsible for conduction disturbances in patients with permanent pacemakers.
  • To identify specific coronary artery pathologies associated with severe conduction abnormalities.

Main Methods:

  • Coronary arteriography was performed on 43 patients with permanent pacemakers and 36 matched controls.
  • Analysis included diameter, stenosis severity, flow assessment, and pathological anatomy, focusing on conduction system territories.
  • Specific attention was paid to lesions in the septal branches and right coronary artery.

Main Results:

  • 27 of 43 pacemaker patients had ischemic heart disease.
  • Conduction disturbances included infranodal (28), sinus nodal (6), AV nodal (4), and complete AV block (5).
  • Pacemaker patients showed significantly different coronary anatomy (compromised septal branches and right coronary artery) compared to controls.

Conclusions:

  • Specific coronary artery anatomy, particularly involving septal branches and the right coronary artery, is associated with severe conduction disturbances requiring pacemakers.
  • The location of coronary lesions, rather than diffuse atherosclerosis, appears critical in causing these conduction abnormalities.
Abstract

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