Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

[Cooperative multicentre study on posttraumatic epilepsy]

M Kobayashi1, T Ohira, M Ishihara

  • 1Department of Neurosurgery, Keio University School of Medicine, Tokyo, Japan.

No to Shinkei = Brain and Nerve
|August 1, 1997
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Cold nuclear matter effects on J/ψ yields as a function of rapidity and nuclear geometry in d+A collisions at sqrt[s(NN)]=200  GeV.

Physical review letters·2011
Same author

Suppression of back-to-back hadron pairs at forward rapidity in d+Au collisions at √s(NN)=200 GeV.

Physical review letters·2011
Same author

Plasmin inhibitor reduces T-cell lymphoid tumor growth by suppressing matrix metalloproteinase-9-dependent CD11b(+)/F4/80(+) myeloid cell recruitment.

Leukemia·2011
Same author

Epstein-Barr virus load for early detection of lymphoproliferative disorder in pediatric renal transplant recipients.

Clinical nephrology·2011
Same author

Cross section and parity-violating spin asymmetries of W± boson production in polarized p + p collisions at sqrt[s] = 500 GeV.

Physical review letters·2011
Same author

Azimuthal anisotropy of π⁰ production in Au+Au collisions at sqrt((s)NN)=200  GeV: path-length dependence of jet quenching and the role of initial geometry.

Physical review letters·2011
Same journal

[Evaluation of cerebral hemodynamics in a patient with middle cerebral artery stenosis by flow-sensitive alternating inversion recovery technique].

No to shinkei = Brain and nerve·2007
Same journal

[Ruptured aneurysm of distal posterior inferior cerebellar artery located at caudal loop].

No to shinkei = Brain and nerve·2007
Same journal

[Bilateral cerebellar infarction caused by intracranial dissection of the vertebral artery after long periods of "Shiatsu"].

No to shinkei = Brain and nerve·2007
Same journal

[Depression following intracerebral hemorrhage and the evaluation of cerebral blood flow by single photon emission tomography].

No to shinkei = Brain and nerve·2007
Same journal

[Study on the distribution of collagen fibers in the bifurcating area of cerebral arterioles of Wistar rats].

No to shinkei = Brain and nerve·2007
Same journal

[Newer antiepileptic drugs].

No to shinkei = Brain and nerve·2007
See all related articles

This study found that brain parenchymal injury and early seizures predict posttraumatic epilepsy (PTE). Anticonvulsant treatment after head injury did not prevent PTE development.

Area of Science:

  • Neuroscience
  • Neurology
  • Trauma Research

Background:

  • Posttraumatic epilepsy (PTE) is a significant concern following head injuries.
  • Identifying predictive factors for PTE and effective prophylactic strategies remains crucial.

Purpose of the Study:

  • To investigate factors influencing the development of posttraumatic epilepsy (PTE).
  • To evaluate the prophylactic efficacy of anticonvulsant medications in preventing PTE after head trauma.

Main Methods:

  • A multicentre prospective study involving 635 head injury patients from April 1994 to April 1996.
  • Anticonvulsants were administered for one month only to patients with brain parenchymal injury.
  • Brain parenchymal injury included subarachnoid hemorrhage, subdural hematoma, contusion, intracerebral hematoma, and diffuse axonal injury.

Related Experiment Videos

Main Results:

  • 14 patients (2.2%) developed PTE, exclusively in those with brain parenchymal injury.
  • Multiple regression analysis identified early epilepsy and brain parenchymal injury as significant predictors of PTE.
  • No significant difference in PTE frequency was observed between the current study's non-anticonvulsant group and a previous untreated retrospective group.

Conclusions:

  • Brain parenchymal injury is a key factor in PTE development.
  • Early epilepsy onset is a predictor for developing PTE.
  • Current evidence suggests anticonvulsant treatment initiated after head injury lacks a prophylactic effect against PTE.