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Soluble adhesion molecules in infants and children undergoing cardiopulmonary bypass

E D Blume1, D P Nelson, K Gauvreau

  • 1Department of Cardiology, Children's Hospital and Harvard Medical School, Boston, Mass 02115, USA.

Circulation
|December 31, 1997
PubMed

Insights

Soluble adhesion molecule levels change after cardiopulmonary bypass (CPB) in children. Higher levels in younger, cyanotic patients with longer CPB indicate greater vascular injury risk, aiding new therapy assessment.

Area of Science:

  • Cardiovascular Surgery
  • Pediatric Cardiology
  • Immunology

Background:

  • Cardiopulmonary bypass (CPB) can cause vascular injury and tissue damage.
  • Leukocyte-endothelial interactions mediated by cell adhesion molecules are key to this injury.

Purpose of the Study:

  • To determine the time course of soluble adhesion molecule levels post-CPB in pediatric patients.
  • To correlate these levels with preoperative variables, intraoperative management, and postoperative outcomes.

Main Methods:

  • Measured plasma concentrations of soluble E-, P-, and L-selectin, ICAM-1, and VCAM-1 in 56 pediatric patients undergoing CPB.
  • Utilized sandwich enzyme-linked immunosorbent assays at multiple time points before, during, and after CPB.
  • Prospectively recorded preoperative, intraoperative, and postoperative data.

Main Results:

  • Soluble adhesion molecule levels initially decreased due to dilution and circuit uptake during CPB.
  • Soluble selectins rose significantly in the first 6 hours post-CPB, returning to baseline by 42 hours.
  • Soluble ICAM-1 and VCAM-1 increased substantially and remained elevated for 42 hours post-CPB.
  • Peak soluble P-selectin correlated with total support time and preoperative cyanosis.
  • Soluble L-selectin levels were inversely associated with longer support/arrest times, intubation duration, cyanosis, and younger age.

Conclusions:

  • Soluble adhesion molecules exhibit a distinct time course following CPB in pediatric patients.
  • Elevated levels are most pronounced in younger, cyanotic patients with longer CPB durations, suggesting higher vascular injury risk.
  • These findings may inform the evaluation of novel therapeutic strategies for CPB-induced vascular injury.
Abstract

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