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Wig-1, a new p53-induced gene encoding a zinc finger protein

S Varmeh-Ziaie1, I Okan, Y Wang

  • 1Microbiology and Tumor Biology Center, Karolinska Institute, Stockholm, Sweden.

Oncogene
|December 24, 1997
PubMed

Insights

Researchers identified a new gene, wig-1, activated by wild type p53. Wig-1 gene expression increases in response to p53 and DNA damage across various mouse tissues.

Area of Science:

  • Molecular Biology
  • Cancer Research
  • Genetics

Background:

  • Wild type p53 protein is a crucial tumor suppressor that regulates cell cycle arrest and apoptosis.
  • The p53-negative J3D mouse T lymphoma line is a model for studying p53 function.
  • Understanding p53-inducible genes is vital for cancer research.

Purpose of the Study:

  • To identify novel genes regulated by wild type p53.
  • To characterize the expression patterns of a newly identified p53-induced gene, wig-1.
  • To investigate the role of wig-1 in response to DNA damage.

Main Methods:

  • Differential display analysis was used to identify p53-induced genes.
  • Quantitative analysis of wig-1 transcript levels in cell lines and mouse tissues.
  • Gamma irradiation was employed to induce DNA damage.

Main Results:

  • A novel p53-induced gene, wig-1, was identified with 7.6 kb and 2.2 kb transcripts.
  • Wig-1 transcripts were upregulated in p53-transfected cells upon wild type p53 induction.
  • Wig-1 induction was observed in irradiated fibroblasts with functional p53 but not in p53-deficient cells.
  • Wig-1 transcripts were induced in multiple mouse organs following gamma irradiation.

Conclusions:

  • Wig-1 is a novel gene transcriptionally regulated by wild type p53.
  • Wig-1 expression is induced by DNA damage in a p53-dependent manner.
  • The WIG-1 protein possesses structural features, including zinc finger motifs, suggesting potential DNA-binding activity.

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