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A multicenter study on insulin-like growth factor-I serum levels in children with chronic inflammatory diseases

R Cimaz1, R Rusconi, B Cesana

  • 1First Pediatric Department, University of Milano, Italy.

Insights

Children and adolescents with connective tissue diseases (CTDs) show lower insulin-like growth factor-I (IGF-I) levels. Inflammation is a key factor, impacting IGF-I levels more than age or steroid treatment.

Area of Science:

  • Pediatric Rheumatology
  • Endocrinology
  • Growth Factors

Background:

  • Connective tissue diseases (CTDs) can affect growth and development in pediatric populations.
  • Insulin-like growth factor-I (IGF-I) plays a crucial role in childhood growth.
  • Understanding IGF-I levels in pediatric CTDs is important for assessing disease impact.

Purpose of the Study:

  • To investigate insulin-like growth factor-I (IGF-I) levels in children and adolescents diagnosed with connective tissue diseases (CTDs).
  • To compare IGF-I levels between pediatric CTD patients and healthy controls.
  • To explore correlations between IGF-I levels and factors such as age, sex, steroid use, and inflammation markers.

Main Methods:

  • A multicenter, cross-sectional study involving 117 pediatric patients with juvenile arthritis and other CTDs, and 78 healthy children.
  • IGF-I serum levels were quantified using radioimmunoassay following acid-ethanol extraction.
  • Statistical analyses included covariance analysis and multiple regression to assess correlations.

Main Results:

  • Mean IGF-I serum levels were significantly lower in patients with CTDs (167.6 ng/ml) compared to controls (214.4 ng/ml).
  • IGF-I levels showed a significant inverse correlation with erythrocyte sedimentation rate (ESR), indicating a link with inflammation.
  • Age and sex were positively correlated with IGF-I levels in patients, while steroid treatment showed no significant correlation.

Conclusions:

  • Pediatric patients with connective tissue diseases exhibit decreased serum IGF-I levels.
  • Inflammatory processes appear to be a significant factor contributing to the reduction in IGF-I levels in this population.
  • Further research may elucidate the precise mechanisms linking inflammation and IGF-I in pediatric CTDs.
Abstract

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