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Non-insulin dependent diabetes mellitus in Mexican-American children

N S Glaser1, K L Jones

  • 1Department of Pediatrics, University of California (UC), Davis, School of Medicine, Sacramento 95817, USA. fzglaser@ucdavis.edu

Insights

Non-insulin-dependent diabetes mellitus (NIDDM) in children is characterized by obesity, acanthosis nigricans, and a strong family history, particularly in Mexican-American youth. Early identification through insulin secretion testing is crucial for appropriate management.

Area of Science:

  • Pediatric Endocrinology
  • Metabolic Disorders
  • Diabetes Mellitus Research

Background:

  • Non-insulin-dependent diabetes mellitus (NIDDM) is increasingly recognized in pediatric populations.
  • Understanding the distinct clinical and metabolic profiles of pediatric NIDDM is essential for accurate diagnosis and management.
  • Previous studies have highlighted the heterogeneity of diabetes in children, necessitating specific characterization of NIDDM subtypes.

Purpose of the Study:

  • To define the clinical and metabolic characteristics of children and adolescents diagnosed with non-insulin-dependent diabetes mellitus (NIDDM).
  • To identify key features that suggest NIDDM in pediatric patients, differentiating them from insulin-dependent diabetes mellitus.
  • To evaluate the prevalence of NIDDM within pediatric diabetes clinics, particularly among specific ethnic groups.

Main Methods:

  • Retrospective review of medical records for 18 children and adolescents meeting NIDDM diagnostic criteria.
  • Inclusion criteria: evidence of sustained endogenous insulin secretion and glycemic control with diet or oral agents for over 2 years.
  • Exclusion criteria: presence of islet cell antibodies or insulin autoantibodies; assessment of clinical features, metabolic markers, and family history.

Main Results:

  • NIDDM represented 8% of all pediatric diabetes cases and 19% of those in Central/South American ancestry; 67% of the study group were Mexican American.
  • Common findings included obesity (67%) and acanthosis nigricans (89%); mean age of onset was 12.8 years.
  • A strong family history of NIDDM was present in 87% of patients, with 47% having affected 3+ generations; mean fasting C-peptide was 1.19 nmol/L.

Conclusions:

  • Pediatric NIDDM is a distinct subset of childhood diabetes, often presenting without ketoacidosis but with acanthosis nigricans, obesity, and a significant family history.
  • Mexican-American children with these clinical features are particularly susceptible to NIDDM.
  • Testing for endogenous insulin secretion is recommended for diabetic children exhibiting these characteristics to ensure appropriate therapeutic strategies.

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