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Community-acquired Acinetobacter pneumonia: a case report

C H Yang¹, K J Chen, C K Wang

¹Department of Internal Medicine, Taipei Municipal Jen-Ai Hospital, Taiwan, ROC.

The Journal of Infection

|February 12, 1998

Summary

A rare case of community-acquired Acinetobacter baumannii pneumonia in a patient with glucose-6-phosphate dehydrogenase deficiency is presented. Despite severe illness including hemolysis and abscess, the patient achieved a full recovery with prompt treatment.

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Area of Science:

Infectious Diseases
Pulmonology
Hematology

Background:

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a genetic disorder that can increase susceptibility to hemolysis.
Acinetobacter baumannii is a bacterium commonly associated with hospital-acquired infections.
Community-acquired pneumonia (CAP) caused by A. baumannii is rare and often severe.

Observation:

A 35-year-old male with G6PD deficiency presented with symptoms of pneumonia, including pleuritic chest pain, fever, and bloody sputum.
Radiography revealed necrotizing pneumonia in the right lower lobe.
The patient developed hypotension, dyspnea, and severe hemolysis.

Findings:

Lung aspiration and sputum cultures identified Acinetobacter baumannii as the causative agent.

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Despite immediate antibiotic therapy, the patient developed an abscess.

The patient experienced a slow but complete recovery after aggressive supportive and antibiotic treatment.

Implications:

This case highlights the potential for A. baumannii to cause severe community-acquired pneumonia, even in immunocompetent individuals.
Prompt diagnosis and aggressive management are crucial for favorable outcomes in such rare cases.
Further research may be needed to understand the specific risk factors and optimal treatment strategies for G6PD-deficient individuals with A. baumannii pneumonia.