Related Experiment Video
Updated: Aug 11, 2026

10:11
Facial Nerve Axotomy in Mice: A Model to Study Motoneuron Response to Injury
Published on: February 23, 2015
Facial nerve paralysis after intratemporal and extratemporal blunt trauma
1Service of Plastic Surgery, Cosme Argerich Hospital, Buenos Aires, Argentina.
The Journal of Craniofacial Surgery
|March 3, 1998
Summary
Blunt facial trauma can cause temporary or permanent facial nerve paralysis. Extratemporal injuries typically result in temporary paralysis with full recovery, while intratemporal injuries may lead to permanent paralysis with uncertain outcomes.
Area of Science:
- Otolaryngology
- Neurology
- Trauma Surgery
Background:
- Definitive facial nerve paralysis from blunt injury is uncommon.
- Facial palsy from closed head trauma usually involves temporal bone fractures.
- Injury to the extratemporal facial nerve trunk is rare, but peripheral branches are more vulnerable.
Observation:
- Comprehensive evaluation including facial muscle function, tear production, taste, and stapedius reflex is crucial.
- Electroneurography and computed tomographic scans are essential for prognosis and fracture localization.
- Prednisolone was used in 4 out of 5 blunt trauma cases, with no surgical interventions.
Findings:
- Extratemporal blunt trauma can cause temporary facial palsy or paresis, with generally complete recovery.
- Intratemporal blunt trauma, with or without fractures, can result in definitive facial nerve paralysis and uncertain recovery.
- Selective peripheral branch injuries (buccal, marginal mandibular) showed complete spontaneous recovery.
Implications:
- Accurate diagnosis differentiating intratemporal vs. extratemporal injury is key for predicting facial nerve recovery.
- Non-surgical management with corticosteroids may be considered for blunt facial nerve trauma.
- Further research is needed to optimize treatment strategies for traumatic facial nerve palsy.
Related Concept Videos
Cranial Nerves: Types Part I
Cranial nerves are responsible for transmitting motor and sensory information between the brain and various parts of the body. There are twelve pairs of cranial nerves, with the first six being essential in sensory perception, motor control, and autonomic functions related to the head and neck.
Olfactory Nerve (Cranial Nerve I)
The olfactory nerve, or cranial nerve I, is unique as it is purely sensory and dedicated to the sense of smell. This nerve originates in the olfactory epithelium of the...
Olfactory Nerve (Cranial Nerve I)
The olfactory nerve, or cranial nerve I, is unique as it is purely sensory and dedicated to the sense of smell. This nerve originates in the olfactory epithelium of the...
Cranial Nerves: Types Part II
Cranial nerves are responsible for transmitting motor and sensory information between the brain and various parts of the body. There are twelve pairs of cranial nerves. While the first six innervate the head and neck, the latter six nerves innervate the head and neck, as well as organs and tissues in the thoracic and abdominal cavities. They facilitate communication, expression, and autonomic control within the human body.
Facial Nerve (Cranial Nerve VII)
Cranial nerve VII, or the facial nerve,...
Facial Nerve (Cranial Nerve VII)
Cranial nerve VII, or the facial nerve,...
Olfactory Receptors: Location and Structure
The process of olfaction, also known as the sense of smell, is a sophisticated chemical response system. The specialized sensory neurons that facilitate this process, known as olfactory receptor neurons, are situated in an upper segment of the nasal cavity, known as the olfactory epithelium. Olfactory sensory neurons are bipolar, with their dendrites extending from the epithelium's apex into the mucus that lines the nasal cavity. Airborne molecules, when inhaled, traverse the olfactory...
Traumatic Brain Injury l: Introduction
DefinitionTraumatic brain injury, or TBI, is a disturbance of normal brain function induced by an external mechanical force, such as a direct blow to the head or a penetrating injury. It can affect both brain structure and function, producing a wide range of clinical outcomes. TBI is a heterogeneous condition, meaning its effects may differ based on the type, location, and severity of the injury.Basis of ClassificationTBI is classified based on severity, injury mechanism, or pathophysiology. In...
Secondary Spinal Cord Injury llI: Pathophysiology
Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...

