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Effect of antimalarial drugs on rat enterocyte mitochondrial phospholipase D activity

M Madesh1, K A Balasubramanian

  • 1Department of Gastrointestinal Sciences, Christian Medical College Hospital, Vellore, India.

Life Sciences
|March 6, 1998
PubMed

Insights

Antimalarial drugs chloroquine and amodiaquine inhibit calcium-stimulated phospholipase D (PLD) in enterocyte mitochondria. These drugs may work by chelating calcium, impacting mitochondrial function.

Area of Science:

  • Mitochondrial biochemistry
  • Pharmacology

Background:

  • Enterocyte mitochondria possess phospholipase D (PLD) activity.
  • PLD can be activated by oxidants, divalent cations, and polyamines.
  • The precise function of mitochondrial PLD remains unclear.

Purpose of the Study:

  • To investigate the effect of antimalarial drugs on mitochondrial PLD activity.
  • To explore the potential mechanism of PLD inhibition by these drugs.

Main Methods:

  • Mitochondrial PLD was activated using oxidants, calcium, or polyamines.
  • The inhibitory effects of chloroquine, amodiaquine, and primaquine on PLD activity were assessed.
  • Dose-dependent inhibition and the impact of varying calcium concentrations were analyzed.

Main Results:

  • Chloroquine and amodiaquine demonstrated dose-dependent inhibition of calcium-stimulated PLD.
  • These drugs did not significantly affect PLD activated by oxidants or polyamines.
  • Increased calcium concentrations reversed the inhibitory effects of chloroquine and amodiaquine.
  • Primaquine showed no significant effect or slight activation of calcium-stimulated PLD.

Conclusions:

  • Chloroquine and amodiaquine likely inhibit mitochondrial PLD by binding to calcium, thus reducing its availability for enzyme activation.
  • This interaction suggests a potential mechanism for how these antimalarial drugs might influence cellular processes involving mitochondrial calcium signaling.

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