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Pseudomonas aeruginosa protease IV produces corneal damage and contributes to bacterial virulence

L S Engel1, J M Hill, J M Moreau

  • 1Department of Microbiology, Immunology, and Parasitology, LSU Eye Center, Louisiana State University Medical Center School of Medicine, New Orleans, USA.

Abstract

Insights

Protease IV directly damages corneas and significantly enhances Pseudomonas virulence in a mouse model of keratitis. This study confirms protease IV

Area of Science:

  • Ophthalmology
  • Microbiology
  • Infectious Diseases

Background:

  • Pseudomonas aeruginosa is a major cause of bacterial keratitis.
  • Protease IV is a secreted virulence factor of Pseudomonas aeruginosa.
  • A mutant deficient in protease IV shows reduced virulence in experimental keratitis.

Purpose of the Study:

  • To determine the corneal toxicity of purified protease IV.
  • To evaluate the ability of protease IV to augment the virulence of protease-IV-deficient Pseudomonas.
  • To elucidate the role of protease IV in the pathogenesis of bacterial keratitis.

Main Methods:

  • Intrastromal injection of purified protease IV (20-200 ng) into mouse corneas.
  • Infection of mouse eyes with protease-IV-deficient Pseudomonas (1000 CFU) plus active or heat-inactivated protease IV.
  • Assessment of ocular disease via slit-lamp examination at multiple time points.
  • Quantification of bacterial load (CFU) in corneas at 27 hours post-infection.

Main Results:

  • Purified protease IV (50-200 ng) induced corneal epithelial defects within 3 hours.
  • Protease IV significantly augmented the virulence of the protease-IV-deficient strain (P < 0.0001).
  • Neither active nor heat-inactivated protease IV affected bacterial growth in the cornea.

Conclusions:

  • Protease IV possesses direct corneal toxicity.
  • Protease IV significantly contributes to Pseudomonas virulence in experimental keratitis.
  • These findings highlight protease IV as a key virulence factor in bacterial keratitis.

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