Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

[Bilateral caudate head infarcts]

N Kuriyama1, Y Yamamoto, I Akiguchi

  • 1Department of Neurology, Kyoto Second Red Cross Hospital.

Rinsho Shinkeigaku = Clinical Neurology
|March 21, 1998
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.

Neuropathology and applied neurobiology·2010
Same author

Cytopathological alterations and therapeutic approaches in Binswanger's disease.

Neuropathology : official journal of the Japanese Society of Neuropathology·2009
Same author

Anti-DARPP32 antibody-immunopositive inclusions in the brain of patients with multiple system atrophy.

Clinical neuropathology·2008
Same author

Age-related changes in barrier function in mouse brain II. Accumulation of serum albumin in the olfactory bulb of SAM mice increased with aging.

Archives of gerontology and geriatrics·2008
Same author

The relationship between 24-hour blood pressure readings, subcortical ischemic lesions and vascular dementia.

Cerebrovascular diseases (Basel, Switzerland)·2005
Same author

CD16+CD57- natural killer cells in multifocal motor neuropathy.

European neurology·2005

Bilateral caudate head infarcts, potentially from cardioembolism, can cause severe abulia and cognitive deficits. This case highlights the significant impact of these specific brain lesions.

Area of Science:

  • Neurology
  • Neuroimaging
  • Vascular Neurology

Background:

  • Caudate head infarcts are rare and can lead to significant neurological deficits.
  • Understanding the vascular supply to the medial striate arteries is crucial for diagnosing such conditions.

Observation:

  • A 67-year-old woman presented with sudden mutism and profound abulia following bilateral caudate head infarcts.
  • Clinical examination revealed apathy, psychomotor slowness, and deficits in spontaneous activity, speech, and attention.
  • Neuroimaging confirmed bilateral caudate head hemorrhagic infarcts extending into the anterior internal capsules, with the left lesion being more extensive.

Findings:

  • The infarcts were likely caused by cardioembolic occlusion affecting the medial striate arteries and Heubner's arteries.

Related Experiment Videos

  • Asymptomatic right internal carotid artery occlusion was noted, along with cardiovascular risk factors including hypertension, diabetes, and atrial fibrillation.
  • Despite treatment and rehabilitation, the patient's condition did not improve, and she died a year later.

    • Implications:

    • Bilateral caudate head infarcts involving the anterior internal capsule can result in severe and persistent abulia.
    • This case underscores the importance of considering cardioembolic sources in patients with bilateral striatal infarcts.
    • Further research into the specific vascular territories and potential contributing anatomical variations is warranted.