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MRI white matter diffusion anisotropy and PET metabolic rate in schizophrenia

M S Buchsbaum1, C Y Tang, S Peled

  • 1Department of Psychiatry, Neuroscience PET Laboratory, Mount Sinai Medical Center, New York, NY 10029-6574, USA.

Neuroreport
|March 25, 1998
PubMed

Insights

Schizophrenia may involve disrupted frontal-striatal brain connections. New MRI scans show reduced white matter integrity in the prefrontal cortex of patients, supporting this theory.

Area of Science:

  • Neuroimaging
  • Psychiatric Neuroscience
  • Brain Connectivity

Background:

  • Frontal-striatal-thalamic circuitry dysfunction is a proposed mechanism in schizophrenia.
  • Previous evidence relied on indirect psychopharmacological and animal model data.
  • Direct assessment of white matter tracts is needed.

Purpose of the Study:

  • To directly investigate white matter integrity in the frontal-striatal pathway in schizophrenia.
  • To examine the relationship between brain structure and metabolic activity in this circuitry.

Main Methods:

  • Employed Diffusion Tensor Imaging (DTI) to assess white matter axon bundles.
  • Utilized high-resolution structural MRI and 18-fluorodeoxyglucose Positron Emission Tomography (PET).
  • Compared five schizophrenia patients with six healthy controls.

Main Results:

  • Significantly lower diffusion anisotropy in the prefrontal cortex white matter of schizophrenia patients.
  • Significantly lower correlation coefficients between prefrontal and striatal metabolic rates in patients.
  • Convergent evidence for diminished fronto-striatal connectivity.

Conclusions:

  • Findings provide direct evidence for impaired white matter connectivity in the frontal-striatal pathway in schizophrenia.
  • Suggests structural and functional deficits contribute to the observed circuitry disturbance.

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