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Identification of restriction barriers in Pasteurella multocida

I C Hoskins1, A J Lax

  • 1Institute for Animal Health, Compton, Berks, UK.

Insights

Antibiotic resistance plasmids in Pasteurella multocida were studied. A specific plasmid, pPM1, showed low transfer into a toxigenic strain, LFB3, but high transfer after recovery, indicating a strain-specific restriction system.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Naturally occurring antibiotic resistance plasmids are significant in bacterial evolution.
  • Pasteurella multocida is an important animal pathogen.
  • Understanding plasmid transfer mechanisms is crucial for controlling antibiotic resistance.

Purpose of the Study:

  • To investigate the transfer of antibiotic resistance plasmids among Pasteurella multocida strains.
  • To characterize the interaction of plasmid pPM1 with the toxigenic P. multocida LFB3 strain.
  • To identify potential restriction-modification systems in P. multocida.

Main Methods:

  • Isolation of antibiotic resistance plasmids from Pasteurella multocida type D.
  • Conjugative and electrotransformation experiments to study plasmid transfer.
  • DNA digestion analysis using restriction enzymes (PstI and DpnI).
  • Plasmid methylation studies.

Main Results:

  • Plasmid pPM1 was successfully transferred to Escherichia coli and some P. multocida isolates.
  • Transfer of pPM1 into toxigenic P. multocida LFB3 occurred at very low frequency.
  • High-frequency transfer of pPM1 to LFB3 was achieved after recovery from electrotransformants.
  • Plasmid DNA from LFB3 was resistant to PstI but sensitive to DpnI, suggesting a PstI restriction system in LFB3.
  • Methylation of pPM1 with PstI methylase increased transformation frequency into LFB3.

Conclusions:

  • The toxigenic Pasteurella multocida LFB3 strain possesses a restriction system targeting PstI sites.
  • This restriction system significantly impacts plasmid DNA transformation efficiency.
  • Understanding these mechanisms is vital for managing antibiotic resistance in P. multocida.

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