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Production of interleukin-12 and expression of its receptors by murine microglia
A Suzumura1, M Sawada, T Takayanagi
1Department of Neurology, Nara Medical University, 840 Shijo-cho, Kashihara, Nara 634, Japan.
Abstract:
Production of interleukin-12 (IL-12) by cultured murine microglia and astrocytes was examined, by means of ELISA to detect heterodimeric p70 and RT-PCR to analyze the expression of mRNA encoding p35 and p40. Microglia, but not astrocytes, produced IL-12 p70 in response to lipopolysaccharide and interferon-gamma. The microglial cell line, Ra2, produced only p40, but not p35, upon above stimulation. Thus, it is possible that some population of microglia induce helper 1 type T cell response via producing IL-12 in the CNS. Microglia were induced to express mRNA encoding IL-12 receptors which were exclusively expressed in activated T and NK cells.
Insights
Murine microglia, but not astrocytes, produce interleukin-12 (IL-12) when stimulated. This suggests microglia may drive T cell responses in the central nervous system (CNS).
Area of Science:
- Neuroimmunology
- Cellular immunology
Background:
- Interleukin-12 (IL-12) is a key cytokine in immune responses.
- Microglia and astrocytes are the primary immune cells in the central nervous system (CNS).
Purpose of the Study:
- To investigate the production of IL-12 by murine microglia and astrocytes.
- To determine the specific subunits (p35 and p40) involved in IL-12 production by these cells.
Main Methods:
- Enzyme-linked immunosorbent assay (ELISA) was used to detect heterodimeric p70.
- Reverse transcription polymerase chain reaction (RT-PCR) was employed to analyze mRNA expression of p35 and p40 subunits.
Main Results:
- Microglia, unlike astrocytes, produced IL-12 p70 in response to lipopolysaccharide and interferon-gamma.
- The microglial cell line Ra2 produced the p40 subunit but not the p35 subunit.
- Microglia expressed mRNA for IL-12 receptors, which are typically found on activated T and NK cells.
Conclusions:
- Certain microglia populations may induce helper 1 (Th1) type T cell responses through IL-12 production within the CNS.
- The findings highlight a potential role for microglia in modulating adaptive immunity within the brain.