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TPM3 deletions cause a hypercontractile congenital muscle stiffness phenotype.

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Polarization from a helix of fluorophores and its relation to that obtained from muscle.

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Reverse actin sliding triggers strong myosin binding that moves tropomyosin.

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Changes in muscle crossbridges when beta, gamma-imido-ATP binds to myosin

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Correction to: The Sydney Heart Bank: improving translational research while eliminating or reducing the use of animal models of human heart disease.

The Sydney Heart Bank: improving translational research while eliminating or reducing the use of animal models of human heart disease.

TPM3 deletions cause a hypercontractile congenital muscle stiffness phenotype.

The properties of glycerol-extracted Longissimus dorsi muscle fibres and myofibrils taken from beef carcasses during post-mortem conditioning.

Polarization from a helix of fluorophores and its relation to that obtained from muscle.

Reverse actin sliding triggers strong myosin binding that moves tropomyosin.

Expanding the Landscape of Disordered Flexible Linkers: A Structural and Computational Framework for DLD dataset assembly.

Codon optimality modulates cellular stress and innate immune responses triggered by exogenous RNAs.

Insights into Genome Ejection by a Therapeutic phiKMV-like Bacteriophage.

In vivo single-molecule tracking of CHD family chromatin remodeler Hrp3 defines the chromatin context-dependent binding dynamics in S. pombe.

Coiled-Coil Domain Kinking Controls Laminin-332 Cleavage by Elastase.

The GCN2-ATF4 Signaling Axis Orchestrates the Hepatic Transcriptional Stress Response to Short-term Sulfur Amino Acid Deprivation.

Impact of Concomitant Grade I Degenerative Spondylolisthesis on Clinical and Radiographic Outcomes Following Biportal Endoscopic versus Microscopic Laminectomy for Lumbar Spinal Stenosis: A Post Hoc Analysis of a Multicenter Randomized Clinical Trial.

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