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Oligomeric properties and DNA binding specificities of repressor isoforms from the Streptomyces bacteriophage phiC31

S E Wilson1, M C Smith

  • 1Department of Genetics, Queens Medical Centre, University Park, Nottingham NG7 2UH, UK.

Insights

The phiC31 bacteriophage repressor gene produces three protein isoforms. These isoforms, particularly the smaller ones, interact and exhibit distinct DNA-binding specificities to conserved inverted repeats, influencing gene regulation.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • The Streptomyces temperate bacteriophage phiC31 repressor gene (gene c) expresses three protein isoforms: 74, 54, and 42 kDa.
  • The 54 and 42 kDa isoforms are sufficient for conferring superinfection immunity, suggesting their critical role in phage regulation.

Purpose of the Study:

  • To investigate the interaction between the 54 and 42 kDa repressor isoforms of bacteriophage phiC31.
  • To determine the DNA-binding specificities of different phiC31 repressor isoforms and their complexes.

Main Methods:

  • Co-purification using Ni-NTA chromatography to study protein interactions.
  • In vitro analysis including chemical crosslinking and gel filtration to identify repressor complex formation (homo- and hetero-tetramers).
  • DNase I footprinting assays to assess DNA-binding activity to conserved inverted repeat (CIR) elements.

Main Results:

  • The 42 kDa native repressor (Nat42) and a 54 kDa histidine-tagged isoform (His54) interact in vivo.
  • Homo- and hetero-tetrameric complexes of the repressor isoforms were observed in vitro.
  • Nat42 showed specific binding to CIR6, while the combined Nat54&42 preparation and His54 bound to multiple CIR elements (CIR3-CIR6) with varying affinities.

Conclusions:

  • The different protein isoforms expressed from the phiC31 repressor gene exhibit distinct DNA-binding specificities.
  • These differential binding properties are analogous to eukaryotic transcription factors and likely contribute to the complex regulation of the phiC31 phage lifecycle.

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