Apoptosis: basic mechanisms and implications for cardiovascular disease

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A C77G point mutation in CD45 exon 4, which is associated with the development of multiple sclerosis and increased susceptibility to HIV-1 infection, is undetectable in Japanese population.

European journal of neurology·2003

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A mouse model of congenital heart disease: cardiac arrhythmias and atrial septal defect caused by haploinsufficiency of the cardiac transcription factor Csx/Nkx2.5.

Cold Spring Harbor symposia on quantitative biology·2003

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The clinical and pathological features of peripheral neuropathy accompanied with HTLV-I associated myelopathy.

Journal of the neurological sciences·2002

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Inhibition of human T-lymphotropic virus type I gene expression by the Streptomyces-derived substance EM2487.

Antiviral chemistry & chemotherapy·2002

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Morphological changes associated with reoxygenation in adult cardiac myocytes.

Journal of cellular and molecular medicine·2002

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Association of CD40 ligand expression on HTLV-I-infected T cells and maturation of dendritic cells.

Scandinavian journal of immunology·2002

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Editors and Editorial Board.

Circulation research·2026

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Cannabis Is Associated With Potentially Proatherogenic Inflammation in Youth.

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WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

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B1a Cell-IgM Axis Protects Against Hypertension by Blunting IFNγ+CD4+T Cells.

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SBK2 Links Cardiac Stress Signaling to Mitochondrial Proteostasis.

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Myeloid Piezo1 as a Brake on Efferocytosis and Cardiac Repair in the Infarcted Heart.

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Related Experiment Videos

Apoptosis: basic mechanisms and implications for cardiovascular disease

Related Concept Videos

A C77G point mutation in CD45 exon 4, which is associated with the development of multiple sclerosis and increased susceptibility to HIV-1 infection, is undetectable in Japanese population.

A mouse model of congenital heart disease: cardiac arrhythmias and atrial septal defect caused by haploinsufficiency of the cardiac transcription factor Csx/Nkx2.5.

The clinical and pathological features of peripheral neuropathy accompanied with HTLV-I associated myelopathy.

Inhibition of human T-lymphotropic virus type I gene expression by the Streptomyces-derived substance EM2487.

Morphological changes associated with reoxygenation in adult cardiac myocytes.

Association of CD40 ligand expression on HTLV-I-infected T cells and maturation of dendritic cells.

Editors and Editorial Board.

Cannabis Is Associated With Potentially Proatherogenic Inflammation in Youth.

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

B1a Cell-IgM Axis Protects Against Hypertension by Blunting IFNγ+CD4+T Cells.

SBK2 Links Cardiac Stress Signaling to Mitochondrial Proteostasis.

Myeloid Piezo1 as a Brake on Efferocytosis and Cardiac Repair in the Infarcted Heart.

Related Experiment Videos

Apoptosis: basic mechanisms and implications for cardiovascular disease

Related Concept Videos

Related Articles

A C77G point mutation in CD45 exon 4, which is associated with the development of multiple sclerosis and increased susceptibility to HIV-1 infection, is undetectable in Japanese population.

A mouse model of congenital heart disease: cardiac arrhythmias and atrial septal defect caused by haploinsufficiency of the cardiac transcription factor Csx/Nkx2.5.

The clinical and pathological features of peripheral neuropathy accompanied with HTLV-I associated myelopathy.

Inhibition of human T-lymphotropic virus type I gene expression by the Streptomyces-derived substance EM2487.

Morphological changes associated with reoxygenation in adult cardiac myocytes.

Association of CD40 ligand expression on HTLV-I-infected T cells and maturation of dendritic cells.

Editors and Editorial Board.

Cannabis Is Associated With Potentially Proatherogenic Inflammation in Youth.

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

B1a Cell-IgM Axis Protects Against Hypertension by Blunting IFNγ+CD4+T Cells.

SBK2 Links Cardiac Stress Signaling to Mitochondrial Proteostasis.

Myeloid Piezo1 as a Brake on Efferocytosis and Cardiac Repair in the Infarcted Heart.