Myocyte cell death in the diseased heart

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Transgenic overexpression of insulin-like growth factor I prevents streptozotocin-induced cardiac contractile dysfunction and beta-adrenergic response in ventricular myocytes.

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Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

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Myocyte cell death in the diseased heart

Related Concept Videos

Resident cardiac stem cells.

Role of stem cells in cardiovascular biology.

Basic research and clinical applications of non-hematopoietic stem cells, 4-5 April 2008, Tubingen, Germany.

Transgenic overexpression of insulin-like growth factor I prevents streptozotocin-induced cardiac contractile dysfunction and beta-adrenergic response in ventricular myocytes.

Cardiomyocyte aging is gender-dependent: the local IGF-1-IGF-1R system.

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

Myeloid Activator Protein-1 Complex Contributes to Salt-Sensitive Hypertension.

Extracellular Vesicles in Cardiovascular Disease: Intercellular Signaling, Liquid Biopsy Biomarkers, and Therapeutic Translation.

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Shifting the Balance: Mitochondrial Heteroplasmy as a Driver of Cardiac Disease.

Natural ERRα Activator Formononetin Ameliorates Anthracycline Cardiotoxicity via Metabolic Improvement.

CDK5RAP3-Mediated Mitochondrial RQC Alleviates Pathological Cardiac Hypertrophy.

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Myocyte cell death in the diseased heart

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