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Vascular endothelial growth factor expression in transient focal cerebral ischemia in the rat
C S Cobbs1, J Chen, D A Greenberg
1Department of Neurosurgery, University of Alabama, Birmingham, USA.
Abstract:
Vascular endothelial growth factor (VEGF) has been implicated in hypoxia-induced angiogenesis in tumors and ischemia. We examined VEGF mRNA and protein expression after occlusion of the middle cerebral artery (MCA) in rats. VEGF mRNA expression studied by in situ hybridization was increased in the ischemic border zone 24 h after 30, 60 or 120 min of focal cerebral ischemia. VEGF protein expression measured by Western blots was also increased in this region 24 and 48 h after ischemia, and VEGF immunocytochemistry localized this increased expression to astroglia. Thus, VEGF is induced after focal cerebral ischemia and could have a role in pathophysiology and recovery in the ischemic border zone.
Insights
Vascular endothelial growth factor (VEGF) increased in rats after focal cerebral ischemia. This suggests VEGF may play a role in the brain
Area of Science:
- Neuroscience
- Molecular Biology
- Pathophysiology
Background:
- Vascular endothelial growth factor (VEGF) is linked to angiogenesis in tumors and ischemia.
- Understanding VEGF's role in cerebral ischemia is crucial for developing therapeutic strategies.
Purpose of the Study:
- To investigate the expression of VEGF mRNA and protein following middle cerebral artery (MCA) occlusion in a rat model.
- To determine the cellular localization of VEGF in the ischemic brain.
Main Methods:
- Focal cerebral ischemia induced by MCA occlusion in rats.
- In situ hybridization for VEGF mRNA detection.
- Western blotting for VEGF protein quantification.
- Immunocytochemistry for cellular localization of VEGF.
Main Results:
- VEGF mRNA expression was elevated in the ischemic border zone 24 hours post-ischemia.
- VEGF protein levels increased in the same region at 24 and 48 hours.
- Immunocytochemistry identified astroglia as the source of increased VEGF expression.
Conclusions:
- VEGF is induced following focal cerebral ischemia in rats.
- The findings suggest a potential role for VEGF in the pathophysiology and recovery processes within the ischemic border zone.