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Transient hypocalcemia with elevated serum parathormone in an infant of a hyperparathyroid mother

E A Ismail1, N al-Shammari, H Nadi

  • 1Department of Paediatrics, Farwaniya Hospital, Kuwait.

Acta Paediatrica Japonica : Overseas Edition
|August 8, 1998
PubMed

Insights

An infant experienced a hypocalcemic fit due to maternal parathyroid adenoma. Treatment with calcium and 1-alpha-hydroxyvitamin D resolved symptoms, suggesting brief end-organ refractoriness.

Area of Science:

  • Pediatric Endocrinology
  • Maternal-Fetal Medicine
  • Endocrinology

Background:

  • Neonatal hypocalcemia can stem from various causes, including maternal conditions.
  • Parathyroid adenoma in mothers can potentially affect infant calcium homeostasis.
  • Secondary causes of end-organ refractoriness must be ruled out in hypocalcemic infants.

Observation:

  • An 80-day-old infant presented with a hypocalcemic fit.
  • The infant's mother had an asymptomatic parathyroid adenoma.
  • Elevated intact parathyroid hormone levels were noted in the infant, without hypomagnesemia, renal failure, or rickets.

Findings:

  • The infant's condition was attributed to end-organ refractoriness, potentially linked to the maternal condition.
  • Treatment with calcium and 1-alpha-hydroxyvitamin D for two weeks resulted in sustained normocalcemia.
  • The infant remained asymptomatic and normocalcemic up to two years of age.

Implications:

  • This case suggests that brief end-organ refractoriness can cause significant hypocalcemia in infants.
  • It highlights the importance of considering maternal parathyroid adenoma as a potential cause of neonatal hypocalcemia.
  • Further research may elucidate the mechanisms of transient end-organ refractoriness in this context.

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