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Molecular typing of multiresistant Streptococcus pneumoniae serogroup 19 in Singapore

T H Koh1, L H Sng, C C Ngan

  • 1Department of Pathology, Singapore General Hospital, Singapore.

Pathology
|December 5, 1998
PubMed

Insights

Penicillin-resistant Streptococcus pneumoniae, particularly multidrug-resistant strains, were prevalent in Singapore. Field-inversion gel electrophoresis (FIGE) identified that these resistant bacteria spread through a limited number of dominant clones.

Area of Science:

  • Microbiology
  • Epidemiology
  • Molecular Biology

Background:

  • Penicillin resistance in Streptococcus pneumoniae is a growing public health concern.
  • In 1995, 25% of clinical Streptococcus pneumoniae isolates at Singapore General Hospital were penicillin-resistant.
  • Multidrug-resistant strains, especially from serogroup 19, were frequently identified.

Purpose of the Study:

  • To investigate the clonal origin of multidrug-resistant serogroup 19 Streptococcus pneumoniae isolates.
  • To determine the genetic relatedness of these resistant strains using molecular typing.

Main Methods:

  • Field-inversion gel electrophoresis (FIGE) was employed for molecular analysis.
  • Chromosomal DNA was digested using restriction enzymes Apal and Smal.
  • Twenty-six isolates, including controls, underwent FIGE analysis.

Main Results:

  • FIGE analysis revealed three distinct clusters of closely related strains.
  • A predominant clone, comprising ten multidrug-resistant serogroup 19 isolates, was identified.
  • This predominant clone also included two control isolates from a different serogroup, suggesting potential cross-contamination or broader dissemination.

Conclusions:

  • The spread of multidrug-resistant serogroup 19 Streptococcus pneumoniae in Singapore is attributed to the dissemination of a small number of successful clones.
  • Molecular typing techniques like FIGE are crucial for understanding bacterial epidemiology and transmission dynamics.
  • These findings highlight the importance of monitoring antibiotic resistance patterns and clonal spread in clinical settings.

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