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The process of ultrastructural changes from nuclei to apoptotic body

T Ihara1, T Yamamoto, M Sugamata

  • 1Department of Pathology, Tochigi Institute of Clinical Pathology, Japan.

Insights

Nivalenol (NIV) induces apoptotic cell death in mouse thymus. This study details the four-stage ultrastructural process of apoptotic body formation, revealing nuclear invagination and crescent-shaped spaces.

Area of Science:

  • Cell Biology
  • Toxicology
  • Pathology

Background:

  • Apoptotic body formation is a key feature of programmed cell death.
  • The precise cellular mechanisms and morphological changes during apoptotic body formation remain incompletely understood.
  • Nivalenol (NIV), a trichothecene mycotoxin, is known to induce cell death.

Purpose of the Study:

  • To investigate the ultrastructural process of apoptotic body formation induced by Nivalenol (NIV).
  • To characterize the morphological changes and cellular pathological processes involved in NIV-induced apoptosis.
  • To elucidate the mechanism of apoptotic body formation through morphological staging.

Main Methods:

  • Induction of apoptotic cell death using Nivalenol (NIV) in mouse thymus.
  • Examination of thymus tissue using electron microscopy at 6, 12, and 18 hours post-administration.
  • Detailed ultrastructural analysis of cellular and nuclear morphology.

Main Results:

  • NIV administration induced apoptotic cell death in the mouse thymus.
  • Early-stage morphological changes included nuclear invagination and pinching off into fragments.
  • Crescent-shaped spaces (CSS) were observed around nuclear envelopes, sometimes containing myelin figures.
  • A four-stage process of apoptotic body formation was identified and characterized.

Conclusions:

  • Nivalenol (NIV) serves as an effective agent to study apoptotic body formation in vivo.
  • The identified four stages provide a framework for understanding the morphological progression of apoptotic body formation.
  • These findings contribute to a clearer understanding of the cellular mechanisms underlying apoptosis.

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