[Atrial thrombosis following the low-energy DC internal countershock of chronic AF: considerations of a clinical

P Palermo1, R Neri, A S Cesario

  • 1Divisione di Cardiologia, Ospedale G.B. Grassi, Roma.

Giornale Italiano Di Cardiologia
|January 15, 1999
PubMed

Insights

Cardioversion for atrial fibrillation can cause new thrombus formation, even without prior clots. This study shows a left atrial appendage thrombus regressed spontaneously after cardioversion, highlighting a potential embolic risk.

Area of Science:

  • Cardiology
  • Cardiac Electrophysiology

Background:

  • Embolism risk persists after cardioversion for atrial arrhythmias like atrial fibrillation, even when pre-existing thrombus is absent.
  • Atrial stunning and de novo thrombus formation post-cardioversion are proposed mechanisms for embolic events.

Observation:

  • A case of chronic atrial fibrillation treated with low-energy internal cardioversion.
  • Recurrent heart failure episodes preceded cardioversion.
  • A de novo left atrial appendage thrombus formed post-cardioversion, monitored via transesophageal echocardiograms.

Findings:

  • The patient developed a new left atrial appendage thrombus after cardioversion.
  • No clinical signs of thromboembolism were observed.
  • The de novo thrombus resolved spontaneously, confirmed by echocardiography.

Implications:

  • This case highlights the potential for de novo thrombus formation after cardioversion, even in the absence of pre-existing thrombus.
  • Spontaneous regression of such thrombi is possible, suggesting a dynamic process.
  • Further research is needed to understand the mechanisms and clinical significance of post-cardioversion de novo thrombus formation.

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