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A Mg(2+)-dependent endonuclease is responsible for internucleosomal DNA fragmentation in human B lymphoblastic IM9

H J Kwon1, D S Kim

  • 1Department of Biochemistry, College of Science, and Bioproducts Research Center, Yonsei University, Seoul, Korea.

Biochemistry and Molecular Biology International
|January 19, 1999
PubMed

Insights

Researchers found a magnesium-dependent endonuclease in human apoptotic cells. This enzyme, localized in the nucleus, is likely responsible for DNA fragmentation during apoptosis.

Area of Science:

  • Biochemistry
  • Molecular Biology
  • Cell Biology

Background:

  • Apoptosis, or programmed cell death, involves DNA fragmentation.
  • The specific enzymes responsible for DNA fragmentation during apoptosis are not fully characterized.
  • Understanding these nucleases is crucial for comprehending cell death pathways.

Purpose of the Study:

  • To identify and characterize a Mg(2+)-dependent endonuclease involved in apoptosis.
  • To investigate the cellular localization and activity of this enzyme.
  • To determine its role in DNA fragmentation in human B lymphoblastic IM9 cells.

Main Methods:

  • Utilized autodigestion and DNA-native-polyacrylamide gel electrophoresis (DNA-native-PAGE) nuclease assay.
  • Assayed endonuclease activity in cell lysates and isolated nuclei.
  • Investigated enzyme activity in cycloheximide-treated cells and apoptotic cells.
  • Isolated the endonuclease via native-PAGE elution.

Main Results:

  • Identified a Mg(2+)-dependent endonuclease activity in IM9 cell lysates and nuclei.
  • Enzyme activity decreased in cell lysates upon cycloheximide treatment but persisted in isolated nuclei of apoptotic cells.
  • The isolated endonuclease converted supercoiled plasmid DNA to linear form.
  • This activity was absent in cycloheximide-treated U937 cells.

Conclusions:

  • A Mg(2+)-dependent endonuclease localized in the nucleus of IM9 cells was identified.
  • This enzyme is likely responsible for the DNA fragmentation observed during apoptosis in these cells.
  • Further research into this endonuclease could reveal new insights into apoptotic mechanisms.

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