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Cellular distribution of phosphorothioate oligonucleotide following intravenous administration in mice

Q Zhao1, R Zhou, J Temsamani

  • 1Hybridon, Inc., Cambridge, MA 02139, USA.

Insights

Fluorescent-labeled oligonucleotides distribute widely in mice, accumulating most in the liver and kidney. This study details oligonucleotide biodistribution and cellular uptake patterns, informing future antisense therapeutic design.

Area of Science:

  • Pharmacology
  • Molecular Biology
  • Biotechnology

Background:

  • Oligonucleotides show therapeutic promise for various diseases.
  • Effective oligonucleotide therapy relies on reaching target cells and organs.
  • Previous studies established phosphorothioate oligonucleotide pharmacokinetics in mice.

Purpose of the Study:

  • To investigate the biodistribution and cellular uptake of fluorescent-labeled oligonucleotides (FITC-oligo) in mice.
  • To extend previous pharmacokinetic studies using a different labeling method.
  • To provide a basis for designing targeted antisense therapeutics.

Main Methods:

  • Administration of 30 mg/kg fluorescent-labeled oligonucleotide (FITC-oligo) to mice.
  • Quantification of fluorescence intensity in various tissues and cells using flow cytometry.
  • Confocal microscopy to visualize intracellular fluorescence in peripheral blood mononuclear cells (PBMC).

Main Results:

  • FITC-oligo was detected in all examined tissues.
  • Highest accumulation observed in liver and kidney, followed by spleen and bone marrow.
  • Peripheral blood mononuclear cells (PBMC) showed very low accumulation.
  • Uptake in PBMC, spleen lymphocytes, and bone marrow cells followed the pattern: monocytes/macrophages > B cells > T cells.
  • Intracellular fluorescence was confirmed in PBMC via confocal microscopy.

Conclusions:

  • Oligonucleotide distribution is tissue-specific, with significant uptake in the liver and kidney.
  • Cellular uptake varies among immune cell types, with monocytes/macrophages showing the highest uptake.
  • These findings support the rationale for developing cell-targeted antisense therapeutics.

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