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Biophysical Journal|April 23, 2013
Synchrony of cardiomyocyte Ca(2+) release is controlled by T-tubule organization, SR Ca(2+) content, and ryanodine receptor Ca(2+) sensitivityLeiv Øyehaug, Kristian Ø Loose, Guro F Jølle, et al.Cardiovascular Research|May 5, 2017
Increased passive stiffness promotes diastolic dysfunction despite improved Ca2+ handling during left ventricular concentric hypertrophyÅsmund T Røe, Jan Magnus Aronsen, Kristine Skårdal, et al.Cardiovascular Research|May 27, 2016
Elevated ventricular wall stress disrupts cardiomyocyte t-tubule structure and calcium homeostasisMichael Frisk, Marianne Ruud, Emil K S Espe, et al.American Journal of Physiology. Heart and Circulatory Physiology|September 21, 2014
Computational modeling of Takotsubo cardiomyopathy: effect of spatially varying β-adrenergic stimulation in the rat left ventricleSander Land, Steven A Niederer, William E Louch, et al.Cardiovascular Research|October 24, 2018
Regional diastolic dysfunction in post-infarction heart failure: role of local mechanical load and SERCA expressionÅsmund T Røe, Marianne Ruud, Emil K Espe, et al.Journal of the American College of Cardiology|January 29, 2021
Etiology-Dependent Impairment of Diastolic Cardiomyocyte Calcium Homeostasis in Heart Failure With Preserved Ejection FractionMichael Frisk, Christopher Le, Xin Shen, et al.Pageof 1