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A Aartsma-Rus

Showing results (1-10 of 16) with videos related to

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Frontiers in Cell and Developmental Biology|January 21, 2025
Histone deacetylase inhibition with givinostat: a multi-targeted mode of action with the potential to halt the pathological cascade of Duchenne muscular dystrophyA Aartsma-Rus
Genes, Brain, and Behavior|May 25, 2016
Cognitive flexibility deficits in a mouse model for the absence of full-length dystrophinE Remmelink, A Aartsma-Rus, A B Smit, et al.
Nature Communications|August 15, 2023
Spatial transcriptomics reveal markers of histopathological changes in Duchenne muscular dystrophy mouse modelsL G M Heezen, T Abdelaal, M van Putten, et al.
Journal of Neuromuscular Diseases|November 19, 2016
Prolonged Ambulation in Duchenne Patients with a Mutation Amenable to Exon 44 SkippingJ C van den Bergen, H B Ginjaar, E H Niks, et al.
Annals of the New York Academy of Sciences|December 6, 2006
Therapeutic modulation of DMD splicing by blocking exonic splicing enhancer sites with antisense oligonucleotidesA Aartsma-Rus, A A M Janson, J A Heemskerk, et al.
Gene Therapy|July 2, 2004
Comparative analysis of antisense oligonucleotide analogs for targeted DMD exon 46 skipping in muscle cellsA Aartsma-Rus, W E Kaman, M Bremmer-Bout, et al.
Iscience|February 11, 2019
Deacetylation Inhibition Reverses PABPN1-Dependent Muscle WastingCyriel S Olie, Muhammad Riaz, Rebecca Konietzny, et al.
Plos One|August 11, 2017
Natural disease history of mouse models for limb girdle muscular dystrophy types 2D and 2FS Pasteuning-Vuhman, K Putker, C L Tanganyika-de Winter, et al.
Plos One|May 16, 2018
Natural disease history of the dy2J mouse model of laminin α2 (merosin)-deficient congenital muscular dystrophyS Pasteuning-Vuhman, K Putker, C L Tanganyika-de Winter, et al.
Human Gene Therapy|September 5, 2007
Comparative analysis of antisense oligonucleotide sequences for targeted skipping of exon 51 during dystrophin pre-mRNA splicing in human muscleV Arechavala-Gomeza, I R Graham, L J Popplewell, et al.
Pageof 2

Showing results (1-10 of 16) with videos related to

Sort By:
Pageof 2
Frontiers in Cell and Developmental Biology|January 21, 2025
Histone deacetylase inhibition with givinostat: a multi-targeted mode of action with the potential to halt the pathological cascade of Duchenne muscular dystrophyA Aartsma-Rus
Genes, Brain, and Behavior|May 25, 2016
Cognitive flexibility deficits in a mouse model for the absence of full-length dystrophinE Remmelink, A Aartsma-Rus, A B Smit, et al.
Nature Communications|August 15, 2023
Spatial transcriptomics reveal markers of histopathological changes in Duchenne muscular dystrophy mouse modelsL G M Heezen, T Abdelaal, M van Putten, et al.
Journal of Neuromuscular Diseases|November 19, 2016
Prolonged Ambulation in Duchenne Patients with a Mutation Amenable to Exon 44 SkippingJ C van den Bergen, H B Ginjaar, E H Niks, et al.
Annals of the New York Academy of Sciences|December 6, 2006
Therapeutic modulation of DMD splicing by blocking exonic splicing enhancer sites with antisense oligonucleotidesA Aartsma-Rus, A A M Janson, J A Heemskerk, et al.
Gene Therapy|July 2, 2004
Comparative analysis of antisense oligonucleotide analogs for targeted DMD exon 46 skipping in muscle cellsA Aartsma-Rus, W E Kaman, M Bremmer-Bout, et al.
Iscience|February 11, 2019
Deacetylation Inhibition Reverses PABPN1-Dependent Muscle WastingCyriel S Olie, Muhammad Riaz, Rebecca Konietzny, et al.
Plos One|August 11, 2017
Natural disease history of mouse models for limb girdle muscular dystrophy types 2D and 2FS Pasteuning-Vuhman, K Putker, C L Tanganyika-de Winter, et al.
Plos One|May 16, 2018
Natural disease history of the dy2J mouse model of laminin α2 (merosin)-deficient congenital muscular dystrophyS Pasteuning-Vuhman, K Putker, C L Tanganyika-de Winter, et al.
Human Gene Therapy|September 5, 2007
Comparative analysis of antisense oligonucleotide sequences for targeted skipping of exon 51 during dystrophin pre-mRNA splicing in human muscleV Arechavala-Gomeza, I R Graham, L J Popplewell, et al.
Pageof 2