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Neurotoxicology|June 9, 2020
Adrenergic β receptor activation reduces amyloid β1-42-mediated intracellular Zn2+ toxicity in dentate granule cells followed by rescuing impairment of dentate gyrus LTPHaruna Tamano, Yudai Ishikawa, Aoi Shioya, et al.
Molecular Neurobiology|February 21, 2018
Novel Defense by Metallothionein Induction Against Cognitive Decline: From Amyloid β1-42-Induced Excess Zn2+ to Functional Zn2+ DeficiencyAtsushi Takeda, Haruna Tamano, Wakana Hashimoto, et al.
Annals of Neurology|February 21, 2013
Ceruloplasmin dysfunction and therapeutic potential for Parkinson diseaseScott Ayton, Peng Lei, James A Duce, et al.
Metallomics : Integrated Biometal Science|January 7, 2020
Characterising the spatial and temporal brain metal profile in a mouse model of tauopathyShalini S Rao, Larissa Lago, Raquel Gonzalez de Vega, et al.
Journal of Alzheimer'S Disease : JAD|September 14, 2020
S-Adenosylmethionine Rescues Cognitive Deficits in the rTg4510 Animal Model by Stabilizing Protein Phosphatase 2A and Reducing Phosphorylated TauLeah C Beauchamp, Xiang M Liu, Amelia Sedjahtera, et al.
Journal of Parkinson'S Disease|November 8, 2021
The Compound ATH434 Prevents Alpha-Synuclein Toxicity in a Murine Model of Multiple System AtrophyDavid I Finkelstein, Jay J Shukla, Robert A Cherny, et al.
ACS Chemical Neuroscience|December 14, 2016
Iron, Copper, and Zinc Concentration in Aβ Plaques in the APP/PS1 Mouse Model of Alzheimer's Disease Correlates with Metal Levels in the Surrounding NeuropilSimon A James, Quentin I Churches, Martin D de Jonge, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 28, 2017
Extracellular Zn2+ Is Essential for Amyloid β1-42-Induced Cognitive Decline in the Normal Brain and Its RescueAtsushi Takeda, Haruna Tamano, Munekazu Tempaku, et al.
Neurobiology of Disease|June 29, 2015
Zinc affects the proteolytic stability of Apolipoprotein E in an isoform-dependent wayHe Xu, Veer B Gupta, Ian J Martins, et al.
Journal of Neurochemistry|February 5, 2019
l-3,4-dihydroxyphenylalanine (l-DOPA) modulates brain iron, dopaminergic neurodegeneration and motor dysfunction in iron overload and mutant alpha-synuclein mouse models of Parkinson's diseaseJessica L Billings, Sarah L Gordon, Tristan Rawling, et al.
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