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A Amalfitano

Showing results (11-20 of 34) with videos related to

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Gene Therapy|June 23, 2001
A modified adenovirus can transfect cochlear hair cells in vivo without compromising cochlear functionA E Luebke, J D Steiger, B L Hodges, et al.
Journal of Innate Immunity|April 9, 2010
TRIF, and TRIF-interacting TLRs differentially modulate several adenovirus vector-induced immune responsesD M Appledorn, S Patial, S Godbehere, et al.
Human Molecular Genetics|October 13, 2000
Mdx mice inducibly expressing dystrophin provide insights into the potential of gene therapy for duchenne muscular dystrophyA Ahmad, M Brinson, B L Hodges, et al.
Journal of Virology|June 1, 1992
Organ- and age-specific replication of polyomavirus in miceJ J Wirth, A Amalfitano, R Gross, et al.
Journal of Virology|March 1, 1990
Low probability of double integration in transformation of nonpermissive cells by polyomavirusS Y Oh, A Amalfitano, K Friderici, et al.
American Journal of Medical Genetics|October 6, 1999
Dubowitz syndrome: a defect in the cholesterol biosynthetic pathway?A Ahmad, A Amalfitano, Y T Chen, et al.
Neuromuscular Disorders : NMD|July 1, 1997
Improved adenoviral vectors for gene therapy of Duchenne muscular dystrophyM A Hauser, A Amalfitano, R Kumar-Singh, et al.
Human Gene Therapy|December 13, 2003
Liver toxicities typically induced by first-generation adenoviral vectors can be reduced by use of E1, E2b-deleted adenoviral vectorsR S Everett, B L Hodges, E Y Ding, et al.
Journal of Virology|January 28, 1998
Production and characterization of improved adenovirus vectors with the E1, E2b, and E3 genes deletedA Amalfitano, M A Hauser, H Hu, et al.
The Journal of Gene Medicine|August 23, 2000
Multiply deleted [E1, polymerase-, and pTP-] adenovirus vector persists despite deletion of the preterminal proteinB L Hodges, D Serra, H Hu, et al.
Pageof 4

Showing results (11-20 of 34) with videos related to

Sort By:
Pageof 4
Gene Therapy|June 23, 2001
A modified adenovirus can transfect cochlear hair cells in vivo without compromising cochlear functionA E Luebke, J D Steiger, B L Hodges, et al.
Journal of Innate Immunity|April 9, 2010
TRIF, and TRIF-interacting TLRs differentially modulate several adenovirus vector-induced immune responsesD M Appledorn, S Patial, S Godbehere, et al.
Human Molecular Genetics|October 13, 2000
Mdx mice inducibly expressing dystrophin provide insights into the potential of gene therapy for duchenne muscular dystrophyA Ahmad, M Brinson, B L Hodges, et al.
Journal of Virology|June 1, 1992
Organ- and age-specific replication of polyomavirus in miceJ J Wirth, A Amalfitano, R Gross, et al.
Journal of Virology|March 1, 1990
Low probability of double integration in transformation of nonpermissive cells by polyomavirusS Y Oh, A Amalfitano, K Friderici, et al.
American Journal of Medical Genetics|October 6, 1999
Dubowitz syndrome: a defect in the cholesterol biosynthetic pathway?A Ahmad, A Amalfitano, Y T Chen, et al.
Neuromuscular Disorders : NMD|July 1, 1997
Improved adenoviral vectors for gene therapy of Duchenne muscular dystrophyM A Hauser, A Amalfitano, R Kumar-Singh, et al.
Human Gene Therapy|December 13, 2003
Liver toxicities typically induced by first-generation adenoviral vectors can be reduced by use of E1, E2b-deleted adenoviral vectorsR S Everett, B L Hodges, E Y Ding, et al.
Journal of Virology|January 28, 1998
Production and characterization of improved adenovirus vectors with the E1, E2b, and E3 genes deletedA Amalfitano, M A Hauser, H Hu, et al.
The Journal of Gene Medicine|August 23, 2000
Multiply deleted [E1, polymerase-, and pTP-] adenovirus vector persists despite deletion of the preterminal proteinB L Hodges, D Serra, H Hu, et al.
Pageof 4