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A Ament

Showing results (131-140 of 148) with videos related to

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Cell Reports|August 20, 2020
Atlas of Transcription Factor Binding Sites from ENCODE DNase Hypersensitivity Data across 27 Tissue TypesCory C Funk, Alex M Casella, Segun Jung, et al.
Molecular Psychiatry|May 13, 2022
Rare variants implicate NMDA receptor signaling and cerebellar gene networks in risk for bipolar disorderNaushaba Hasin, Lace M Riggs, Tatyana Shekhtman, et al.
Molecular Psychiatry|June 30, 2018
Rediscovering the value of families for psychiatric genetics researchDavid C Glahn, Vishwajit L Nimgaonkar, Henriette Raventós, et al.
Biorxiv : the Preprint Server for Biology|September 18, 2025
Inhibition of autophagy-lysosomal function exacerbates microglial and monocyte lipid metabolism reprograming and dysfunction after brain injuryAmir A Mehrabani-Tabari, Nivedita Hegdekar, Brian R Herb, et al.
Scientific Reports|March 29, 2017
Corrigendum: Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt<sup>Q111/+</sup> model of Huntington's diseaseRobert M Bragg, Sydney R Coffey, Rory M Weston, et al.
Research Square|November 24, 2025
Inhibition of autophagy-lysosomal function exacerbates microglial and monocyte lipid metabolism reprograming and dysfunction after brain injuryAmir A Mehrabani-Tabari, Nivedita Hegdekar, Brian R Herb, et al.
Human Molecular Genetics|March 24, 2017
High resolution time-course mapping of early transcriptomic, molecular and cellular phenotypes in Huntington's disease CAG knock-in mice across multiple genetic backgroundsSeth A Ament, Jocelynn R Pearl, Andrea Grindeland, et al.
Scientific Reports|February 9, 2017
Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt<sup>Q111/+</sup> model of Huntington's diseaseRobert M Bragg, Sydney R Coffey, Rory M Weston, et al.
Nature Neuroscience|March 26, 2026
NeMO Analytics: a compendium of transcriptomic data for the exploration of neocortical developmentShreyash Sonthalia, Brian Herb, Ricky S Adkins, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 3, 2015
Rare variants in neuronal excitability genes influence risk for bipolar disorderSeth A Ament, Szabolcs Szelinger, Gustavo Glusman, et al.
Pageof 15

Showing results (131-140 of 148) with videos related to

Sort By:
Pageof 15
Cell Reports|August 20, 2020
Atlas of Transcription Factor Binding Sites from ENCODE DNase Hypersensitivity Data across 27 Tissue TypesCory C Funk, Alex M Casella, Segun Jung, et al.
Molecular Psychiatry|May 13, 2022
Rare variants implicate NMDA receptor signaling and cerebellar gene networks in risk for bipolar disorderNaushaba Hasin, Lace M Riggs, Tatyana Shekhtman, et al.
Molecular Psychiatry|June 30, 2018
Rediscovering the value of families for psychiatric genetics researchDavid C Glahn, Vishwajit L Nimgaonkar, Henriette Raventós, et al.
Biorxiv : the Preprint Server for Biology|September 18, 2025
Inhibition of autophagy-lysosomal function exacerbates microglial and monocyte lipid metabolism reprograming and dysfunction after brain injuryAmir A Mehrabani-Tabari, Nivedita Hegdekar, Brian R Herb, et al.
Scientific Reports|March 29, 2017
Corrigendum: Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt<sup>Q111/+</sup> model of Huntington's diseaseRobert M Bragg, Sydney R Coffey, Rory M Weston, et al.
Research Square|November 24, 2025
Inhibition of autophagy-lysosomal function exacerbates microglial and monocyte lipid metabolism reprograming and dysfunction after brain injuryAmir A Mehrabani-Tabari, Nivedita Hegdekar, Brian R Herb, et al.
Human Molecular Genetics|March 24, 2017
High resolution time-course mapping of early transcriptomic, molecular and cellular phenotypes in Huntington's disease CAG knock-in mice across multiple genetic backgroundsSeth A Ament, Jocelynn R Pearl, Andrea Grindeland, et al.
Scientific Reports|February 9, 2017
Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt<sup>Q111/+</sup> model of Huntington's diseaseRobert M Bragg, Sydney R Coffey, Rory M Weston, et al.
Nature Neuroscience|March 26, 2026
NeMO Analytics: a compendium of transcriptomic data for the exploration of neocortical developmentShreyash Sonthalia, Brian Herb, Ricky S Adkins, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 3, 2015
Rare variants in neuronal excitability genes influence risk for bipolar disorderSeth A Ament, Szabolcs Szelinger, Gustavo Glusman, et al.
Pageof 15