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Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|July 16, 2008
Targeting beta-amyloid pathology in Alzheimer's disease with Abeta immunotherapyRoger M Nitsch, Christoph HockNeuroreport|July 4, 2001
Compartmentalized tau hyperphosphorylation and increased levels of kinases in transgenic miceJ Götz, R M NitschPediatric Surgery International|September 24, 2013
Metabolic consequences of sigmoidocystoplasty in childrenU Beseghi, J M Guys, V Dibenedetto, et al.Neuro-Degenerative Diseases|August 16, 2006
Clinical observations with AN-1792 using TAPIR analysesChristoph Hock, Roger M NitschBiological Chemistry Hoppe-Seyler|July 1, 1990
Separation of granule subpopulations in human polymorphonuclear leukocytesM Nitsch, D Gabrijelcic, H TschescheNeurobiology of Aging|January 5, 2002
Genetics of interleukin 6: implications for Alzheimer's diseaseA Papassotiropoulos, C Hock, R M NitschIn Vivo (Athens, Greece)|June 27, 2007
Head and neck cancer triggers increased IL-6 production of CD34+ stem cells from human cord bloodStefan M Nitsch, Ralph Pries, Barbara WollenbergActa Neuropathologica|March 19, 2016
Tau pathology-dependent remodelling of cerebral arteries precedes Alzheimer's disease-related microvascular cerebral amyloid angiopathyMario Merlini, Debora Wanner, Roger M NitschPlos One|September 23, 2020
Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo systemChristian Tackenberg, Luka Kulic, Roger M NitschPageof 28