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Current Alzheimer Research|November 20, 2012
Brief but chronic increase in allopregnanolone cause accelerated AD pathology differently in two mouse modelsSara K Bengtsson, Maja Johansson, Torbjorn Backstrom, et al.
Neurobiology of Disease|July 6, 2010
Vascular response to acetazolamide decreases as a function of age in the arcA beta mouse model of cerebral amyloidosisFelicitas L Princz-Kranz, Thomas Mueggler, Marlen Knobloch, et al.
Annals of the New York Academy of Sciences|February 24, 2001
In vivo analysis of wild-type and FTDP-17 tau transgenic miceJ Götz, R Barmettler, A Ferrari, et al.
The European Journal of Neuroscience|June 26, 2001
Oligodendroglial tau filament formation in transgenic mice expressing G272V tauJ Götz, M Tolnay, R Barmettler, et al.
Journal of Molecular Biology|March 16, 2011
The Osaka FAD mutation E22Δ leads to the formation of a previously unknown type of amyloid β fibrils and modulates Aβ neurotoxicityOxana Yu Ovchinnikova, Verena H Finder, Ivana Vodopivec, et al.
Annals of the New York Academy of Sciences|January 22, 2005
BACE1 overexpression regulates amyloid precursor protein cleavage and interaction with the ShcA adapterEmanuela Repetto, Claudio Russo, Valentina Venezia, et al.
Brain Pathology (Zurich, Switzerland)|September 1, 2020
Distinct changes in all major components of the neurovascular unit across different neuropathological stages of Alzheimer's diseaseTunahan Kirabali, Ruslan Rust, Serena Rigotti, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 21, 2014
A single-trial estimation of the feedback-related negativity and its relation to BOLD responses in a time-estimation taskMichael P I Becker, Alexander M Nitsch, Wolfgang H R Miltner, et al.
Genetic Counseling (Geneva, Switzerland)|March 23, 2007
Anophthalmos with limb anomalies (Waardenburg opththalmo-acromelic syndrome): report of a new Italian case with renal anomaly and reviewL Garavelli, S Pedori, R Dal Zotto, et al.
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