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Cancer Cells (Cold Spring Harbor, N.Y. : 1989)|September 1, 1991
Isoprenoid modification and plasma membrane association: critical factors for ras oncogenicityC J Der, A D CoxFASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 1, 1993
Emerging concepts in the Ras superfamily of GTP-binding proteinsG M Bokoch, C J DerCell|January 1, 1983
Altered gene products are associated with activation of cellular rasK genes in human lung and colon carcinomasC J Der, G M CooperInternational Journal of Cancer|October 15, 1980
Alterations in the extracellular matrix organization associated with the reexpression of tumorigenicity in human cell hybridsC J Der, E J StanbridgeCell|January 17, 1986
Biological and biochemical properties of human rasH genes mutated at codon 61C J Der, T Finkel, G M CooperCell|May 1, 1984
Activation of ras genes in human tumors does not affect localization, modification, or nucleotide binding properties of p21T Finkel, C J Der, G M CooperCurrent Opinion in Cell Biology|August 1, 1992
Structure and biological effects of lipid modifications on proteinsM Chow, C J Der, J E BussThe Journal of Biological Chemistry|September 29, 2000
Ras inactivation of the retinoblastoma pathway by distinct mechanisms in NIH 3T3 fibroblast and RIE-1 epithelial cellsK Pruitt, R G Pestell, C J DerSeminars in Cancer Biology|August 1, 1992
Prenoids and palmitate: lipids that control the biological activity of Ras proteinsK Kato, C J Der, J E BussPageof 17