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A C FISHER

Showing results (81-90 of 91) with videos related to

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Leukemia|September 26, 2014
Clonal evolution revealed by whole genome sequencing in a case of primary myelofibrosis transformed to secondary acute myeloid leukemiaE K Engle, D A C Fisher, C A Miller, et al.
Leukemia|December 24, 2016
Mass cytometry analysis reveals hyperactive NF Kappa B signaling in myelofibrosis and secondary acute myeloid leukemiaD A C Fisher, O Malkova, E K Engle, et al.
Blood Advances|October 13, 2021
Pevonedistat targets malignant cells in myeloproliferative neoplasms in vitro and in vivo via NFκB pathway inhibitionTim Kong, Angelo B A Laranjeira, Taylor B Collins, et al.
Nature Communications|March 24, 2023
IMC-Denoise: a content aware denoising pipeline to enhance Imaging Mass CytometryPeng Lu, Karolyn A Oetjen, Diane E Bender, et al.
Cell Communication and Signaling : CCS|December 16, 2025
A proteogenomic gene signature defines prognostic subgroups highlighting PI3K/AKT/mTOR signaling pathway as a therapeutic vulnerability in myeloid malignanciesFan He, Shuyang Lin, Bei Gao, et al.
Clinical Immunology (Orlando, Fla.)|November 5, 2018
A Markov Multi-State model of lupus nephritis urine biomarker panel dynamics in children: Predicting changes in disease activityE M D Smith, A Eleuteri, B Goilav, et al.
Cancer Cell|November 14, 2018
JARID2 Functions as a Tumor Suppressor in Myeloid Neoplasms by Repressing Self-Renewal in Hematopoietic Progenitor CellsHamza Celik, Won Kyun Koh, Ashley C Kramer, et al.
Nature Cancer|December 29, 2022
DUSP6 mediates resistance to JAK2 inhibition and drives leukemic progressionTim Kong, Angelo B A Laranjeira, Kangning Yang, et al.
Cancer Discovery|July 1, 2021
A Humanized Animal Model Predicts Clonal Evolution and Therapeutic Vulnerabilities in Myeloproliferative NeoplasmsHamza Celik, Ethan Krug, Christine R Zhang, et al.
Blood|June 25, 2026
Galectin-1 Fuels Monocyte Hyperinflammation and Represents a Novel Therapeutic Target in Myeloproliferative NeoplasmsFan He, Shuyang Lin, Tim Kong, et al.
Pageof 10

Showing results (81-90 of 91) with videos related to

Sort By:
Pageof 10
Leukemia|September 26, 2014
Clonal evolution revealed by whole genome sequencing in a case of primary myelofibrosis transformed to secondary acute myeloid leukemiaE K Engle, D A C Fisher, C A Miller, et al.
Leukemia|December 24, 2016
Mass cytometry analysis reveals hyperactive NF Kappa B signaling in myelofibrosis and secondary acute myeloid leukemiaD A C Fisher, O Malkova, E K Engle, et al.
Blood Advances|October 13, 2021
Pevonedistat targets malignant cells in myeloproliferative neoplasms in vitro and in vivo via NFκB pathway inhibitionTim Kong, Angelo B A Laranjeira, Taylor B Collins, et al.
Nature Communications|March 24, 2023
IMC-Denoise: a content aware denoising pipeline to enhance Imaging Mass CytometryPeng Lu, Karolyn A Oetjen, Diane E Bender, et al.
Cell Communication and Signaling : CCS|December 16, 2025
A proteogenomic gene signature defines prognostic subgroups highlighting PI3K/AKT/mTOR signaling pathway as a therapeutic vulnerability in myeloid malignanciesFan He, Shuyang Lin, Bei Gao, et al.
Clinical Immunology (Orlando, Fla.)|November 5, 2018
A Markov Multi-State model of lupus nephritis urine biomarker panel dynamics in children: Predicting changes in disease activityE M D Smith, A Eleuteri, B Goilav, et al.
Cancer Cell|November 14, 2018
JARID2 Functions as a Tumor Suppressor in Myeloid Neoplasms by Repressing Self-Renewal in Hematopoietic Progenitor CellsHamza Celik, Won Kyun Koh, Ashley C Kramer, et al.
Nature Cancer|December 29, 2022
DUSP6 mediates resistance to JAK2 inhibition and drives leukemic progressionTim Kong, Angelo B A Laranjeira, Kangning Yang, et al.
Cancer Discovery|July 1, 2021
A Humanized Animal Model Predicts Clonal Evolution and Therapeutic Vulnerabilities in Myeloproliferative NeoplasmsHamza Celik, Ethan Krug, Christine R Zhang, et al.
Blood|June 25, 2026
Galectin-1 Fuels Monocyte Hyperinflammation and Represents a Novel Therapeutic Target in Myeloproliferative NeoplasmsFan He, Shuyang Lin, Tim Kong, et al.
Pageof 10