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Journal of Bioenergetics and Biomembranes|November 7, 2001
Mitochondrial genetic control of assembly and function of complex I in mammalian cellsA ChomynCurrent Genetics|August 1, 1989
cDNA of the 24 kDa subunit of the bovine respiratory chain NADH dehydrogenase: high sequence conservation in mammals and tissue-specific and growth-dependent expressionA Chomyn, S S LaiBiochimica Et Biophysica Acta|May 24, 1995
Complementation and segregation behavior of disease-causing mitochondrial DNA mutations in cellular model systemsG Attardi, M Yoneda, A ChomynNucleic Acids Research|February 25, 1981
Alignment of the amino terminal amino acid sequence of human cytochrome c oxidase subunits I and II with the sequence of their putative mRNAsA Chomyn, M W Hunkapiller, G AttardiNature Genetics|May 1, 1995
MtDNA mutation in MERRF syndrome causes defective aminoacylation of tRNA(Lys) and premature translation terminationJ A Enriquez, A Chomyn, G AttardiThe Journal of Clinical Investigation|April 1, 1992
Zidovudine induces molecular, biochemical, and ultrastructural changes in rat skeletal muscle mitochondriaW Lewis, B Gonzalez, A Chomyn, et al.Nucleic Acids Research|January 16, 1999
Search for differences in post-transcriptional modification patterns of mitochondrial DNA-encoded wild-type and mutant human tRNALys and tRNALeu(UUR)M Helm, C Florentz, A Chomyn, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 7, 1997
Myoclonic epilepsy and ragged red fibers (MERRF) syndrome: selective vulnerability of CNS neurons does not correlate with the level of mitochondrial tRNAlys mutation in individual neuronal isolatesL Zhou, A Chomyn, G Attardi, et al.The Journal of Biological Chemistry|March 5, 1986
Antibodies against the COOH-terminal undecapeptide of subunit II, but not those against the NH2-terminal decapeptide, immunoprecipitate the whole human cytochrome c oxidase complexP Mariottini, A Chomyn, R F Doolittle, et al.Proceedings of the National Academy of Sciences of the United States of America|December 1, 1992
Marked replicative advantage of human mtDNA carrying a point mutation that causes the MELAS encephalomyopathyM Yoneda, A Chomyn, A Martinuzzi, et al.Pageof 3