Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

A Devin

Showing results (11-20 of 29) with videos related to

Pageof 3
Sort By:
European Journal of Biochemistry|July 17, 2001
Allosteric activation of pyruvate kinase via NAD+ in rat liver cellsA Devin, V Nogueira, X Leverve, et al.
European Journal of Biochemistry|October 1, 1996
Mechanisms of inhibition and uncoupling of respiration in isolated rat liver mitochondria by the general anesthetic 2,6-diisopropylphenolM Rigoulet, A Devin, N Avéret, et al.
Immunity|May 5, 2000
The distinct roles of TRAF2 and RIP in IKK activation by TNF-R1: TRAF2 recruits IKK to TNF-R1 while RIP mediates IKK activationA Devin, A Cook, Y Lin, et al.
Molecular and Cellular Biology|May 22, 2001
The alpha and beta subunits of IkappaB kinase (IKK) mediate TRAF2-dependent IKK recruitment to tumor necrosis factor (TNF) receptor 1 in response to TNFA Devin, Y Lin, S Yamaoka, et al.
Molecular and Cellular Biochemistry|September 24, 1998
Oxidative phosphorylation in intact hepatocytes: quantitative characterization of the mechanisms of change in efficiency and cellular consequencesX Leverve, B Sibille, A Devin, et al.
The Journal of Biological Chemistry|October 2, 2001
Mitochondrial respiratory chain adjustment to cellular energy demandV Nogueira, M Rigoulet, M A Piquet, et al.
The Journal of Biological Chemistry|April 1, 2000
Disruption of hsp90 function results in degradation of the death domain kinase, receptor-interacting protein (RIP), and blockage of tumor necrosis factor-induced nuclear factor-kappaB activationJ Lewis, A Devin, A Miller, et al.
Molecular and Cellular Biology|August 25, 2000
The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinaseY Lin, A Devin, A Cook, et al.
Biochimica Et Biophysica Acta|September 30, 1996
Effect of polyunsaturated fatty acids deficiency on oxidative phosphorylation in rat liver mitochondriaE M Fontaine, M Moussa, A Devin, et al.
Biochimica Et Biophysica Acta|August 7, 1998
Flux-force relationships in intact cells: a helpful tool for understanding the mechanism of oxidative phosphorylation alterations?M Rigoulet, A Devin, P Espié, et al.
Pageof 3

Showing results (11-20 of 29) with videos related to

Sort By:
Pageof 3
European Journal of Biochemistry|July 17, 2001
Allosteric activation of pyruvate kinase via NAD+ in rat liver cellsA Devin, V Nogueira, X Leverve, et al.
European Journal of Biochemistry|October 1, 1996
Mechanisms of inhibition and uncoupling of respiration in isolated rat liver mitochondria by the general anesthetic 2,6-diisopropylphenolM Rigoulet, A Devin, N Avéret, et al.
Immunity|May 5, 2000
The distinct roles of TRAF2 and RIP in IKK activation by TNF-R1: TRAF2 recruits IKK to TNF-R1 while RIP mediates IKK activationA Devin, A Cook, Y Lin, et al.
Molecular and Cellular Biology|May 22, 2001
The alpha and beta subunits of IkappaB kinase (IKK) mediate TRAF2-dependent IKK recruitment to tumor necrosis factor (TNF) receptor 1 in response to TNFA Devin, Y Lin, S Yamaoka, et al.
Molecular and Cellular Biochemistry|September 24, 1998
Oxidative phosphorylation in intact hepatocytes: quantitative characterization of the mechanisms of change in efficiency and cellular consequencesX Leverve, B Sibille, A Devin, et al.
The Journal of Biological Chemistry|October 2, 2001
Mitochondrial respiratory chain adjustment to cellular energy demandV Nogueira, M Rigoulet, M A Piquet, et al.
The Journal of Biological Chemistry|April 1, 2000
Disruption of hsp90 function results in degradation of the death domain kinase, receptor-interacting protein (RIP), and blockage of tumor necrosis factor-induced nuclear factor-kappaB activationJ Lewis, A Devin, A Miller, et al.
Molecular and Cellular Biology|August 25, 2000
The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinaseY Lin, A Devin, A Cook, et al.
Biochimica Et Biophysica Acta|September 30, 1996
Effect of polyunsaturated fatty acids deficiency on oxidative phosphorylation in rat liver mitochondriaE M Fontaine, M Moussa, A Devin, et al.
Biochimica Et Biophysica Acta|August 7, 1998
Flux-force relationships in intact cells: a helpful tool for understanding the mechanism of oxidative phosphorylation alterations?M Rigoulet, A Devin, P Espié, et al.
Pageof 3