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Gene Therapy|June 15, 2001
Wild-type p53 gene transfer inhibits neointima formation in human saphenous vein by modulation of smooth muscle cell migration and induction of apoptosisS J George, G D Angelini, M C Capogrossi, et al.Gene Therapy|February 4, 1999
Gene transfer of tissue inhibitor of metalloproteinase-2 inhibits metalloproteinase activity and neointima formation in human saphenous veinsS J George, A H Baker, G D Angelini, et al.Current Gene Therapy|March 23, 2004
Promoters and control elements: designing expression cassettes for gene therapyE D Papadakis, S A Nicklin, A H Baker, et al.The Journal of Clinical Investigation|April 29, 1998
Divergent effects of tissue inhibitor of metalloproteinase-1, -2, or -3 overexpression on rat vascular smooth muscle cell invasion, proliferation, and death in vitro. TIMP-3 promotes apoptosisA H Baker, A B Zaltsman, S J George, et al.Gene Therapy|March 26, 2016
The relevance of coagulation factor X protection of adenoviruses in human seraM R Duffy, A Doszpoly, G Turner, et al.Biology of Reproduction|December 22, 1998
Rat amnion type IV collagen composition and metabolism: implications for membrane breakdownH Lei, R Kalluri, E E Furth, et al.British Journal of Cancer|April 3, 1999
Inhibition of invasion and induction of apoptotic cell death of cancer cell lines by overexpression of TIMP-3A H Baker, S J George, A B Zaltsman, et al.Gene Therapy|August 5, 2004
Dual targeting of gene delivery by genetic modification of adenovirus serotype 5 fibers and cell-selective transcriptional controlL M Work, N Ritchie, S A Nicklin, et al.Matrix Biology : Journal of the International Society for Matrix Biology|December 1, 1996
Development of recombinant adenoviruses that drive high level expression of the human metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 and -2 genes: characterization of their infection into rabbit smooth muscle cells and human MCF-7 adenocarcinoma cellsA H Baker, G W Wilkinson, R M Hembry, et al.The Biochemical Journal|April 1, 1996
Divergent regulation by growth factors and cytokines of 95 kDa and 72 kDa gelatinases and tissue inhibitors or metalloproteinases-1, -2, and -3 in rabbit aortic smooth muscle cellsR P Fabunmi, A H Baker, E J Murray, et al.Pageof 8