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European Heart Journal|February 25, 2005
Intense metabolic control by means of insulin in patients with diabetes mellitus and acute myocardial infarction (DIGAMI 2): effects on mortality and morbidityK Malmberg, L Rydén, H Wedel, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 15, 2002
Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of EuropeI Juhan-Vague, P E Morange, H Aubert, et al.
Atherosclerosis|September 19, 2012
LDL oxidative modification and carotid atherosclerosis: results of a multicenter studyK Nyyssönen, S Kurl, J Karppi, et al.
Journal of Internal Medicine|December 21, 2013
The HLA locus contains novel foetal susceptibility alleles for congenital heart block with significant paternal influenceS Meisgen, T Östberg, S Salomonsson, et al.
Journal of Molecular Medicine (Berlin, Germany)|November 11, 2008
Genetic variants of TNFSF4 and risk for carotid artery disease and strokeP S Olofsson, L A Söderström, C Jern, et al.
Journal of Thrombosis and Haemostasis : JTH|November 25, 2003
The plasminogen activator inhibitor-1 -675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH studyI Juhan-Vague, P E Morange, C Frere, et al.
Diabetologia|February 19, 2009
A common variant in PNPLA3, which encodes adiponutrin, is associated with liver fat content in humansA Kotronen, L E Johansson, L M Johansson, et al.
Journal of Internal Medicine|July 23, 2017
Integrative studies implicate matrix metalloproteinase-12 as a culprit gene for large-artery atherosclerotic strokeH Mahdessian, L Perisic Matic, M Lengquist, et al.
Atherosclerosis|June 1, 2005
EPCR Ser219Gly: elevated sEPCR, prothrombin F1+2, risk for coronary heart disease, and increased sEPCR shedding in vitroH Ireland, C J Konstantoulas, J A Cooper, et al.
Journal of Internal Medicine|June 29, 2019
Subclinical atherosclerosis and its progression are modulated by PLIN2 through a feed-forward loop between LXR and autophagyP Saliba-Gustafsson, M Pedrelli, K Gertow, et al.
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