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Annals of Neurology|January 1, 1990
Opioid and nonopioid mechanisms may contribute to dynorphin's pathophysiological actions in spinal cord injuryA I FadenBrain Research|January 15, 1990
Competitive and non-competitive NMDA antagonists limit dynorphin A-induced rat hindlimb paralysisR Bakshi, A I FadenBritish Journal of Pharmacology|August 1, 1983
Hypothalamic regulation of the cardiovascular and respiratory systems: role of specific opiate receptorsA I Faden, G FeuersteinLife Sciences|November 15, 1982
Differential cardiovascular effects of mu, delta and kappa opiate agonists at discrete hypothalamic sites in the anesthetized ratG Feuerstein, A I FadenThe Journal of Pharmacology and Experimental Therapeutics|June 1, 1992
Platelet-activating factor reduces spinal cord blood flow and causes behavioral deficits after intrathecal administration in rats through a specific receptor mechanismA I Faden, P HaltTrends in Pharmacological Sciences|January 1, 1992
Pharmacological strategies in CNS traumaA I Faden, S SalzmanNeuroscience Letters|March 2, 1990
Blockade of the glycine modulatory site of NMDA receptors modifies dynorphin-induced behavioral effectsR Bakshi, A I FadenNeuropeptides|December 1, 1984
Cardiovascular effects of dynorphin A-(1-8), dynorphin A-(1-13) and dynorphin A-(1-17) microinjected into the preoptic medialis nucleus of the ratG Feuerstein, A I FadenJournal of Neurotrauma|January 1, 1990
Edema development and ion changes in rat spinal cord after impact trauma: injury dose-response studiesM Lemke, A I FadenJournal of Neuroscience Research|May 1, 1988
Traumatic spinal cord injury in rats causes increases in tissue thromboxane but not peptidoleukotrienesP Demediuk, A I FadenPageof 23