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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 25, 1999
Mitochondrial depolarization is not required for neuronal apoptosisA J Krohn, T Wahlbrink, J H PrehnThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 8, 1998
Staurosporine-induced apoptosis of cultured rat hippocampal neurons involves caspase-1-like proteases as upstream initiators and increased production of superoxide as a main downstream effectorA J Krohn, E Preis, J H PrehnNeuroscience|February 28, 2001
Up-regulation of Bcl-xL in response to subtoxic beta-amyloid: role in neuronal resistance against apoptotic and oxidative injuryC M Luetjens, S Lankiewicz, N T Bui, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 26, 2000
Delayed mitochondrial dysfunction in excitotoxic neuron death: cytochrome c release and a secondary increase in superoxide productionC M Luetjens, N T Bui, B Sengpiel, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 28, 2001
Dissipation of potassium and proton gradients inhibits mitochondrial hyperpolarization and cytochrome c release during neural apoptosisM Poppe, C Reimertz, H Düssmann, et al.The Journal of Biological Chemistry|May 29, 2000
Activation of calpain I converts excitotoxic neuron death into a caspase-independent cell deathS Lankiewicz, C Marc Luetjens, N Truc Bui, et al.Pageof 1