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A J L Clark

Showing results (1-10 of 25) with videos related to

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Best Practice & Research. Clinical Endocrinology & Metabolism|April 22, 2018
Pathophysiology of melanocortin receptors and their accessory proteinsT V Novoselova, L F Chan, A J L Clark
Hormone Research|December 7, 2007
Familial glucocorticoid deficiency: advances in the molecular understanding of ACTH actionL F Chan, A J L Clark, L A Metherell
European Journal of Endocrinology|September 26, 2007
Clinical and biological phenotype of a patient with familial glucocorticoid deficiency type 2 caused by a mutation of melanocortin 2 receptor accessory proteinH Rumié, L A Metherell, A J L Clark, et al.
Neuroscience|February 1, 2005
Identification of the sites of expression of triple A syndrome mRNA in the rat using in situ hybridisationH L Storr, A J L Clark, J V Priestley, et al.
The Journal of Endocrinology|February 19, 2013
Melanocortin receptor accessory proteins in adrenal gland physiology and beyondT V Novoselova, D Jackson, D C Campbell, et al.
Endocrine Development|February 9, 2013
ACTH resistance: genes and mechanismsE Meimaridou, C R Hughes, J Kowalczyk, et al.
European Journal of Endocrinology|November 12, 2009
Isolated Addison's disease is unlikely to be caused by mutations in MC2R, MRAP or STAR, three genes responsible for familial glucocorticoid deficiencyR P Dias, L F Chan, L A Metherell, et al.
Endocrine Research|January 18, 2003
Agonist activated adrenocorticotropin receptor internalizes via a clathrin-mediated G protein receptor kinase dependent mechanismA H Baig, F M Swords, M Szaszák, et al.
The Journal of Endocrinology|July 6, 2002
Failed export of the adrenocorticotrophin receptor from the endoplasmic reticulum in non-adrenal cells: evidence in support of a requirement for a specific adrenal accessory factorL A Noon, J M Franklin, P J King, et al.
The Journal of Clinical Endocrinology and Metabolism|April 30, 2010
Missense mutations in the melanocortin 2 receptor accessory protein that lead to late onset familial glucocorticoid deficiency type 2C R Hughes, T T Chung, A M Habeb, et al.
Pageof 3

Showing results (1-10 of 25) with videos related to

Sort By:
Pageof 3
Best Practice & Research. Clinical Endocrinology & Metabolism|April 22, 2018
Pathophysiology of melanocortin receptors and their accessory proteinsT V Novoselova, L F Chan, A J L Clark
Hormone Research|December 7, 2007
Familial glucocorticoid deficiency: advances in the molecular understanding of ACTH actionL F Chan, A J L Clark, L A Metherell
European Journal of Endocrinology|September 26, 2007
Clinical and biological phenotype of a patient with familial glucocorticoid deficiency type 2 caused by a mutation of melanocortin 2 receptor accessory proteinH Rumié, L A Metherell, A J L Clark, et al.
Neuroscience|February 1, 2005
Identification of the sites of expression of triple A syndrome mRNA in the rat using in situ hybridisationH L Storr, A J L Clark, J V Priestley, et al.
The Journal of Endocrinology|February 19, 2013
Melanocortin receptor accessory proteins in adrenal gland physiology and beyondT V Novoselova, D Jackson, D C Campbell, et al.
Endocrine Development|February 9, 2013
ACTH resistance: genes and mechanismsE Meimaridou, C R Hughes, J Kowalczyk, et al.
European Journal of Endocrinology|November 12, 2009
Isolated Addison's disease is unlikely to be caused by mutations in MC2R, MRAP or STAR, three genes responsible for familial glucocorticoid deficiencyR P Dias, L F Chan, L A Metherell, et al.
Endocrine Research|January 18, 2003
Agonist activated adrenocorticotropin receptor internalizes via a clathrin-mediated G protein receptor kinase dependent mechanismA H Baig, F M Swords, M Szaszák, et al.
The Journal of Endocrinology|July 6, 2002
Failed export of the adrenocorticotrophin receptor from the endoplasmic reticulum in non-adrenal cells: evidence in support of a requirement for a specific adrenal accessory factorL A Noon, J M Franklin, P J King, et al.
The Journal of Clinical Endocrinology and Metabolism|April 30, 2010
Missense mutations in the melanocortin 2 receptor accessory protein that lead to late onset familial glucocorticoid deficiency type 2C R Hughes, T T Chung, A M Habeb, et al.
Pageof 3